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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2001-4-3
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pubmed:abstractText |
Increased intracellular calcium accumulation is known to potentiate ischemic injury. Whether endogenous calcium-binding proteins can attenuate this injury has not been clearly established, and existing data are conflicting. Calbindin D28K (CaBP) is one such intracellular calcium buffer. We investigated whether CaBP overexpression is neuroprotective against transient focal cerebral ischemia.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
1524-4628
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
32
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1028-35
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:11283407-Animals,
pubmed-meshheading:11283407-Brain,
pubmed-meshheading:11283407-Calcium-Binding Protein, Vitamin D-Dependent,
pubmed-meshheading:11283407-Cell Count,
pubmed-meshheading:11283407-Cell Survival,
pubmed-meshheading:11283407-Corpus Striatum,
pubmed-meshheading:11283407-Disease Models, Animal,
pubmed-meshheading:11283407-Gene Expression,
pubmed-meshheading:11283407-Gene Therapy,
pubmed-meshheading:11283407-Genes, Reporter,
pubmed-meshheading:11283407-Genetic Vectors,
pubmed-meshheading:11283407-Ischemic Attack, Transient,
pubmed-meshheading:11283407-Male,
pubmed-meshheading:11283407-Microinjections,
pubmed-meshheading:11283407-Neurons,
pubmed-meshheading:11283407-Rats,
pubmed-meshheading:11283407-Rats, Sprague-Dawley
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pubmed:year |
2001
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pubmed:articleTitle |
Calbindin d28k overexpression protects striatal neurons from transient focal cerebral ischemia.
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pubmed:affiliation |
Department of Neurosurgery, Stanford University, California, USA. yenari@stanford.edu
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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