Linked Life Data

11275628

Source:http://linkedlifedata.com/resource/pubmed/id/11275628

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2001-3-29
pubmed:abstractText
The etiology of minimal-change nephrotic syndrome (MCNS) is poorly understood. It has been proposed that cell-mediated immunity and T-cell activation are key features of this glomerular disease. Interleukin (IL)-18, a novel interferon-gamma-stimulating factor, may act as an important effector molecule involved in various immune responses. To our knowledge, very little is known about the involvement of IL-18 in NCNS. The aim here was to define further the involvement of IL-18 in MCNS.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
0250-8095
pubmed:author
pubmed:issnType
Print
pubmed:volume
21
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
20-7
pubmed:dateRevised
2007-2-14
pubmed:meshHeading
pubmed:articleTitle
Augmented interleukin-18 production by peripheral blood monocytes in patients with minimal-change nephrotic syndrome.
pubmed:affiliation
Department of Medical Technology, College of Medical Sciences, Saitama Prefectural University, Koshigaya, Japan.
pubmed:publicationType
Journal Article