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| pubmed-article:10982776 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
| pubmed-article:10982776 | lifeskim:mentions | umls-concept:C0034721 | lld:lifeskim |
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| pubmed-article:10982776 | lifeskim:mentions | umls-concept:C0030274 | lld:lifeskim |
| pubmed-article:10982776 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
| pubmed-article:10982776 | lifeskim:mentions | umls-concept:C0135208 | lld:lifeskim |
| pubmed-article:10982776 | lifeskim:mentions | umls-concept:C1444748 | lld:lifeskim |
| pubmed-article:10982776 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:10982776 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:10982776 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:10982776 | pubmed:dateCreated | 2000-9-29 | lld:pubmed |
| pubmed-article:10982776 | pubmed:abstractText | Tumor necrosis factor (TNF)-alpha contributes to the development of acute pancreatitis. Because TNF-alpha is involved in the control of apoptosis, we studied its interaction with the pancreatic apoptotic pathway. | lld:pubmed |
| pubmed-article:10982776 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10982776 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10982776 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:10982776 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10982776 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:10982776 | pubmed:issn | 0016-5085 | lld:pubmed |
| pubmed-article:10982776 | pubmed:author | pubmed-author:DagornJ CJC | lld:pubmed |
| pubmed-article:10982776 | pubmed:author | pubmed-author:MalkaDD | lld:pubmed |
| pubmed-article:10982776 | pubmed:author | pubmed-author:BödekerHH | lld:pubmed |
| pubmed-article:10982776 | pubmed:author | pubmed-author:VerrandoPP | lld:pubmed |
| pubmed-article:10982776 | pubmed:author | pubmed-author:IovannaJ LJL | lld:pubmed |
| pubmed-article:10982776 | pubmed:author | pubmed-author:DusettiN JNJ | lld:pubmed |
| pubmed-article:10982776 | pubmed:author | pubmed-author:OrtizE MEM | lld:pubmed |
| pubmed-article:10982776 | pubmed:author | pubmed-author:VasseurSS | lld:pubmed |
| pubmed-article:10982776 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10982776 | pubmed:volume | 119 | lld:pubmed |
| pubmed-article:10982776 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10982776 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10982776 | pubmed:pagination | 816-28 | lld:pubmed |
| pubmed-article:10982776 | pubmed:dateRevised | 2010-2-8 | lld:pubmed |
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| pubmed-article:10982776 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:10982776 | pubmed:articleTitle | Tumor necrosis factor alpha triggers antiapoptotic mechanisms in rat pancreatic cells through pancreatitis-associated protein I activation. | lld:pubmed |
| pubmed-article:10982776 | pubmed:affiliation | Laboratoire de Recherche de Physiologie et Pathologie Digestives, INSERM Unité 315, Marseille, France. | lld:pubmed |
| pubmed-article:10982776 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10982776 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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