| pubmed-article:10916092 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10916092 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:10916092 | lifeskim:mentions | umls-concept:C0010453 | lld:lifeskim |
| pubmed-article:10916092 | lifeskim:mentions | umls-concept:C0069676 | lld:lifeskim |
| pubmed-article:10916092 | lifeskim:mentions | umls-concept:C0227651 | lld:lifeskim |
| pubmed-article:10916092 | lifeskim:mentions | umls-concept:C0033634 | lld:lifeskim |
| pubmed-article:10916092 | lifeskim:mentions | umls-concept:C1120843 | lld:lifeskim |
| pubmed-article:10916092 | lifeskim:mentions | umls-concept:C0086597 | lld:lifeskim |
| pubmed-article:10916092 | lifeskim:mentions | umls-concept:C1514485 | lld:lifeskim |
| pubmed-article:10916092 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:10916092 | pubmed:dateCreated | 2000-9-7 | lld:pubmed |
| pubmed-article:10916092 | pubmed:abstractText | We previously reported that hypoxia induces the proliferation of cultured mesangial cells mediated by the stimulation of intracellular calcium and the activation of protein kinase C (PKC). In the present study, we examined the roles of mesangial cell specific growth factors (platelet-derived growth factor and endothelin-1) and osteopontin (OPN) in hypoxia-induced proliferation of mesangial cells. In addition, we determined the effect of hypoxia on p38 mitogen-activated protein (MAP) kinase activity and the roles of both PKC and p38 MAP kinase in hypoxia-induced alterations in OPN and mesangial cell growth. | lld:pubmed |
| pubmed-article:10916092 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10916092 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10916092 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:10916092 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:10916092 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10916092 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:10916092 | pubmed:issn | 0085-2538 | lld:pubmed |
| pubmed-article:10916092 | pubmed:author | pubmed-author:SahaiAA | lld:pubmed |
| pubmed-article:10916092 | pubmed:author | pubmed-author:BatlleDD | lld:pubmed |
| pubmed-article:10916092 | pubmed:author | pubmed-author:SodhiC PCP | lld:pubmed |
| pubmed-article:10916092 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10916092 | pubmed:volume | 58 | lld:pubmed |
| pubmed-article:10916092 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10916092 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10916092 | pubmed:pagination | 691-700 | lld:pubmed |
| pubmed-article:10916092 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:10916092 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:10916092 | pubmed:articleTitle | Osteopontin mediates hypoxia-induced proliferation of cultured mesangial cells: role of PKC and p38 MAPK. | lld:pubmed |
| pubmed-article:10916092 | pubmed:affiliation | Division of Nephrology and Hypertension, Northwestern University Medical School, Chicago, IL 60611, USA. | lld:pubmed |
| pubmed-article:10916092 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10916092 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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