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rdf:type |
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|
lifeskim:mentions |
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pubmed:issue |
5
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pubmed:dateCreated |
2000-9-14
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pubmed:abstractText |
The Zucker fatty fa/fa rat develops hyperinsulinaemia, insulin-resistance and severe obesity as a result of a homozygous mutation in the leptin receptor gene. The aim was to characterise the metabolic defect(s) in hepatocytes from fa/fa rats.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
0012-186X
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
43
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
589-97
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:10855534-Animals,
pubmed-meshheading:10855534-Female,
pubmed-meshheading:10855534-Glucokinase,
pubmed-meshheading:10855534-Gluconeogenesis,
pubmed-meshheading:10855534-Glycogen,
pubmed-meshheading:10855534-Glycolysis,
pubmed-meshheading:10855534-Liver,
pubmed-meshheading:10855534-Male,
pubmed-meshheading:10855534-Mannitol,
pubmed-meshheading:10855534-Obesity,
pubmed-meshheading:10855534-Rats,
pubmed-meshheading:10855534-Rats, Wistar,
pubmed-meshheading:10855534-Rats, Zucker
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pubmed:year |
2000
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pubmed:articleTitle |
Impaired glycogen synthesis in hepatocytes from Zucker fatty fa/fa rats: the role of increased phosphorylase activity.
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pubmed:affiliation |
Department of Diabetes and Metabolism, The Medical School, University of Newcastle upon Tyne, UK.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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