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pubmed-article:10817564 | lifeskim:mentions | umls-concept:C0017296 | lld:lifeskim |
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pubmed-article:10817564 | lifeskim:mentions | umls-concept:C0016030 | lld:lifeskim |
pubmed-article:10817564 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
pubmed-article:10817564 | lifeskim:mentions | umls-concept:C1550315 | lld:lifeskim |
pubmed-article:10817564 | lifeskim:mentions | umls-concept:C0521447 | lld:lifeskim |
pubmed-article:10817564 | lifeskim:mentions | umls-concept:C1518440 | lld:lifeskim |
pubmed-article:10817564 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
pubmed-article:10817564 | lifeskim:mentions | umls-concept:C1274040 | lld:lifeskim |
pubmed-article:10817564 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
pubmed-article:10817564 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:10817564 | pubmed:dateCreated | 2000-6-1 | lld:pubmed |
pubmed-article:10817564 | pubmed:abstractText | Tumor necrosis factor alpha (TNFalpha) increases the survival and proliferation of human rheumatoid arthritis (RA) cell lines. These experiments were designed to determine if inhibition of nuclear factor kappaB (NF-kappaB) nuclear translocation leads to increased apoptosis of TNFalpha-treated human RA cell lines. | lld:pubmed |
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pubmed-article:10817564 | pubmed:language | eng | lld:pubmed |
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pubmed-article:10817564 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:10817564 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:10817564 | pubmed:month | May | lld:pubmed |
pubmed-article:10817564 | pubmed:issn | 0004-3591 | lld:pubmed |
pubmed-article:10817564 | pubmed:author | pubmed-author:LieTT | lld:pubmed |
pubmed-article:10817564 | pubmed:author | pubmed-author:MountzJ DJD | lld:pubmed |
pubmed-article:10817564 | pubmed:author | pubmed-author:ZhangXX | lld:pubmed |
pubmed-article:10817564 | pubmed:author | pubmed-author:YangPP | lld:pubmed |
pubmed-article:10817564 | pubmed:author | pubmed-author:ZhangH GHG | lld:pubmed |
pubmed-article:10817564 | pubmed:author | pubmed-author:ZhouTT | lld:pubmed |
pubmed-article:10817564 | pubmed:author | pubmed-author:CurielD TDT | lld:pubmed |
pubmed-article:10817564 | pubmed:author | pubmed-author:HuangNN | lld:pubmed |
pubmed-article:10817564 | pubmed:author | pubmed-author:BilbanMM | lld:pubmed |
pubmed-article:10817564 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:10817564 | pubmed:volume | 43 | lld:pubmed |
pubmed-article:10817564 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:10817564 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:10817564 | pubmed:pagination | 1094-105 | lld:pubmed |
pubmed-article:10817564 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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pubmed-article:10817564 | pubmed:year | 2000 | lld:pubmed |
pubmed-article:10817564 | pubmed:articleTitle | Gene therapy that inhibits nuclear translocation of nuclear factor kappaB results in tumor necrosis factor alpha-induced apoptosis of human synovial fibroblasts. | lld:pubmed |
pubmed-article:10817564 | pubmed:affiliation | University of Alabama at Birmingham, 35294, USA. | lld:pubmed |
pubmed-article:10817564 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:10817564 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:10817564 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
pubmed-article:10817564 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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