Source:http://linkedlifedata.com/resource/pubmed/id/10809426
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| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:issue |
4
|
| pubmed:dateCreated |
2000-7-13
|
| pubmed:abstractText |
CsA-induced endothelial dysfunction and CsA-induced hypertension have been attributed to CsA effects on the endothelial-derived factors controlling vasomotor tone, but the mechanisms responsible are unclear. Endothelial nitric oxide (NO) is known to maintain a state of basal vasodilation and recently a NO mediated counterregulatory mechanism protective from CsA-induced vasoconstriction has been suggested.
|
| pubmed:language |
eng
|
| pubmed:journal | |
| pubmed:citationSubset |
IM
|
| pubmed:chemical | |
| pubmed:status |
MEDLINE
|
| pubmed:month |
Apr
|
| pubmed:issn |
0301-0430
|
| pubmed:author | |
| pubmed:issnType |
Print
|
| pubmed:volume |
53
|
| pubmed:owner |
NLM
|
| pubmed:authorsComplete |
Y
|
| pubmed:pagination |
suppl 6-7
|
| pubmed:dateRevised |
2005-11-17
|
| pubmed:meshHeading |
pubmed-meshheading:10809426-Humans,
pubmed-meshheading:10809426-Hypertension, Renal,
pubmed-meshheading:10809426-Kidney Transplantation,
pubmed-meshheading:10809426-Nitric Oxide,
pubmed-meshheading:10809426-Nitric Oxide Synthase,
pubmed-meshheading:10809426-Oxidative Stress,
pubmed-meshheading:10809426-Urothelium
|
| pubmed:year |
2000
|
| pubmed:articleTitle |
Oxidative stress and nitric oxide system in post-transplant hypertension.
|
| pubmed:affiliation |
Department of Clinical and Experimental Medicine, Clinica Medica 4, University of Padova, Italy.
|
| pubmed:publicationType |
Journal Article
|