Source:http://linkedlifedata.com/resource/pubmed/id/10197914
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
1
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pubmed:dateCreated |
1999-7-16
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pubmed:abstractText |
Rats with a neurotoxic lesion of the amygdala central nucleus (CN) in one hemisphere and a 192 immunoglobulin G (192IgG)-saporin lesion of cholinergic neurons in the contralateral substantia innominata/nucleus basalis (SI/nBM) failed to show the enhanced attentional processing of a conditioned stimulus (CS) observed in sham-operated rats when that CS's predictive value was altered. Performance of these asymmetrically lesioned rats was poorer than that of rats with a unilateral lesion of either structure or with a symmetrical lesion of both structures in the same hemisphere. These results implicate connections between the CN and SI/nBM in the incremental attentional processing of CSs, extending previous research that has shown similar effects of bilateral lesions of either the CN or the SI/nBM.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
0735-7044
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
113
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
143-51
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:10197914-Amygdala,
pubmed-meshheading:10197914-Animals,
pubmed-meshheading:10197914-Attention,
pubmed-meshheading:10197914-Brain Mapping,
pubmed-meshheading:10197914-Cholinergic Fibers,
pubmed-meshheading:10197914-Conditioning, Classical,
pubmed-meshheading:10197914-Dominance, Cerebral,
pubmed-meshheading:10197914-Male,
pubmed-meshheading:10197914-Neural Pathways,
pubmed-meshheading:10197914-Rats,
pubmed-meshheading:10197914-Rats, Long-Evans,
pubmed-meshheading:10197914-Substantia Innominata
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pubmed:year |
1999
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pubmed:articleTitle |
Disconnection of the amygdala central nucleus and substantia innominata/nucleus basalis disrupts increments in conditioned stimulus processing in rats.
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pubmed:affiliation |
Department of Psychology, Johns Hopkins University, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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