10021355

Source:http://linkedlifedata.com/resource/pubmed/id/10021355

Download in:

Switch to

Custom View

Named Graph Language Inference

Statements in which the resource exists as a subject.
Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0029016, umls-concept:C0079427, umls-concept:C0694768, umls-concept:C1325410
pubmed:issue	2
pubmed:dateCreated	1999-3-23
pubmed:abstractText	The adenomatous polyposis coli (APC) tumor suppressor protein is inactivated by mutations in the majority of colorectal cancers. A recent study has revealed that alterations in the APC signaling pathway can result in the transcriptional activation of the c-MYC gene.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9107782
pubmed:citationSubset	IM
pubmed:status	MEDLINE
pubmed:month	Jan
pubmed:issn	0960-9822
pubmed:author	pubmed-author:DangC VCV, pubmed-author:FearonE RER
pubmed:issnType	Print
pubmed:day	28
pubmed:volume	9
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	R62-5
pubmed:dateRevised	2008-11-21
pubmed:meshHeading	pubmed-meshheading:10021355-Genes, APC, pubmed-meshheading:10021355-Genes, myc, pubmed-meshheading:10021355-Humans, pubmed-meshheading:10021355-Mutation, pubmed-meshheading:10021355-Oncogenes, pubmed-meshheading:10021355-Signal Transduction, pubmed-meshheading:10021355-Transcriptional Activation
pubmed:year	1999
pubmed:articleTitle	Cancer genetics: tumor suppressor meets oncogene.
pubmed:affiliation	Division of Molecular Medicine and Genetics, Department of Internal Medicine, Human Genetics, University of Michigan Cancer Center, Ann Arbor, 48109-0638 USA. fearon@umich.edu
pubmed:publicationType	Journal Article, Review