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rdf:type |
|
|
lifeskim:mentions |
|
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pubmed:issue |
2
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pubmed:dateCreated |
1999-4-15
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pubmed:abstractText |
The evidence that transforming growth factor-beta (TGF-beta) is a key mediator in the pathogenesis of fibrotic diseases is now supported by several lines of investigation. This evidence provides a certain base for targeting TGF-beta as an antifibrotic agent.
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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|
pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Feb
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pubmed:issn |
0085-2538
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pubmed:author |
pubmed-author:AkagiYY,
pubmed-author:AndoYY,
pubmed-author:BorderW AWA,
pubmed-author:HoriMM,
pubmed-author:ImaiEE,
pubmed-author:IsakaYY,
pubmed-author:KanedaYY,
pubmed-author:NAVAG CGC,
pubmed-author:NobleN ANA,
pubmed-author:SudoTT,
pubmed-author:TsujieMM
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pubmed:issnType |
Print
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pubmed:volume |
55
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pubmed:owner |
NLM
|
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pubmed:authorsComplete |
Y
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pubmed:pagination |
465-75
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:9987071-Animals,
pubmed-meshheading:9987071-Chimera,
pubmed-meshheading:9987071-Extracellular Matrix,
pubmed-meshheading:9987071-Feasibility Studies,
pubmed-meshheading:9987071-Gene Expression,
pubmed-meshheading:9987071-Gene Therapy,
pubmed-meshheading:9987071-Glomerular Mesangium,
pubmed-meshheading:9987071-Glomerulonephritis,
pubmed-meshheading:9987071-Immunoglobulin Fc Fragments,
pubmed-meshheading:9987071-Immunoglobulin G,
pubmed-meshheading:9987071-Male,
pubmed-meshheading:9987071-Rats,
pubmed-meshheading:9987071-Rats, Sprague-Dawley,
pubmed-meshheading:9987071-Receptors, Transforming Growth Factor beta
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pubmed:year |
1999
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|
pubmed:articleTitle |
Gene therapy by transforming growth factor-beta receptor-IgG Fc chimera suppressed extracellular matrix accumulation in experimental glomerulonephritis.
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pubmed:affiliation |
First Department of Medicine, Osaka University School of Medicine, Japan.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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