rdf:type |
|
lifeskim:mentions |
umls-concept:C0003483,
umls-concept:C0005823,
umls-concept:C0012963,
umls-concept:C0021467,
umls-concept:C0021469,
umls-concept:C0036945,
umls-concept:C0132555,
umls-concept:C0205217,
umls-concept:C0205373,
umls-concept:C0871261,
umls-concept:C1704632,
umls-concept:C1706817,
umls-concept:C1880177,
umls-concept:C2911692
|
pubmed:issue |
2
|
pubmed:dateCreated |
1999-2-10
|
pubmed:abstractText |
To determine whether inotropic responses to the beta-adrenergic agonist dobutamine are potentiated by systemic inhibition of nitric oxide synthase (NOS) with the L-arginine analogue N omega-nitro-L-arginine (L-NNA), and to establish to what extent any observed responses are related to the increase in aortic blood pressure accompanying systemic NOS inhibition.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Nov
|
pubmed:issn |
0008-6363
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
40
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
282-9
|
pubmed:dateRevised |
2007-11-15
|
pubmed:meshHeading |
pubmed-meshheading:9893721-Adrenergic beta-Agonists,
pubmed-meshheading:9893721-Analysis of Variance,
pubmed-meshheading:9893721-Animals,
pubmed-meshheading:9893721-Aorta,
pubmed-meshheading:9893721-Blood Pressure,
pubmed-meshheading:9893721-Dobutamine,
pubmed-meshheading:9893721-Dose-Response Relationship, Drug,
pubmed-meshheading:9893721-Heart Rate,
pubmed-meshheading:9893721-Hemodynamics,
pubmed-meshheading:9893721-Ligation,
pubmed-meshheading:9893721-Myocardial Contraction,
pubmed-meshheading:9893721-Nitric Oxide Synthase,
pubmed-meshheading:9893721-Nitroarginine,
pubmed-meshheading:9893721-Sheep,
pubmed-meshheading:9893721-Stimulation, Chemical,
pubmed-meshheading:9893721-Ventricular Function, Left,
pubmed-meshheading:9893721-Ventricular Pressure
|
pubmed:year |
1998
|
pubmed:articleTitle |
Increased aortic blood pressure contributes to potentiated dobutamine inotropic responses after systemic NO synthase inhibition in sheep.
|
pubmed:affiliation |
Institute of Reproduction and Development, Monash University, Clayton, Victoria, Australia.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|