| pubmed-article:9885877 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9885877 | lifeskim:mentions | umls-concept:C0205054 | lld:lifeskim |
| pubmed-article:9885877 | lifeskim:mentions | umls-concept:C0376618 | lld:lifeskim |
| pubmed-article:9885877 | lifeskim:mentions | umls-concept:C0600437 | lld:lifeskim |
| pubmed-article:9885877 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:9885877 | lifeskim:mentions | umls-concept:C0037533 | lld:lifeskim |
| pubmed-article:9885877 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:9885877 | pubmed:dateCreated | 1999-3-29 | lld:pubmed |
| pubmed-article:9885877 | pubmed:abstractText | Endotoxin rapidly inhibits the activity of the constitutive endothelial nitric oxide synthase (ecNOS); this precedes the production of NO from inducible NOS (iNOS). This leaves a period in early endotoxaemia with a supposed scarcity of NO. The present study was conducted to examine the effects of external supplementation of NO on liver microcirculation and function. | lld:pubmed |
| pubmed-article:9885877 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9885877 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9885877 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9885877 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:9885877 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9885877 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9885877 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9885877 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9885877 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9885877 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9885877 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:9885877 | pubmed:issn | 0342-4642 | lld:pubmed |
| pubmed-article:9885877 | pubmed:author | pubmed-author:MessmerKK | lld:pubmed |
| pubmed-article:9885877 | pubmed:author | pubmed-author:AasenA OAO | lld:pubmed |
| pubmed-article:9885877 | pubmed:author | pubmed-author:LilleaasenPP | lld:pubmed |
| pubmed-article:9885877 | pubmed:author | pubmed-author:LeidererRR | lld:pubmed |
| pubmed-article:9885877 | pubmed:author | pubmed-author:CorteC DCD | lld:pubmed |
| pubmed-article:9885877 | pubmed:author | pubmed-author:GundersenYY | lld:pubmed |
| pubmed-article:9885877 | pubmed:author | pubmed-author:DörgerMM | lld:pubmed |
| pubmed-article:9885877 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9885877 | pubmed:volume | 24 | lld:pubmed |
| pubmed-article:9885877 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9885877 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9885877 | pubmed:pagination | 1257-63 | lld:pubmed |
| pubmed-article:9885877 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:meshHeading | pubmed-meshheading:9885877-... | lld:pubmed |
| pubmed-article:9885877 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9885877 | pubmed:articleTitle | The nitric oxide donor sodium nitroprusside protects against hepatic microcirculatory dysfunction in early endotoxaemia. | lld:pubmed |
| pubmed-article:9885877 | pubmed:affiliation | Institute for Surgical Research, Rikshospitalet, University of Oslo, Norway. | lld:pubmed |
| pubmed-article:9885877 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9885877 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:9885877 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:9885877 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:9885877 | lld:pubmed |
