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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
12
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pubmed:dateCreated |
1999-3-29
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pubmed:abstractText |
Endotoxin rapidly inhibits the activity of the constitutive endothelial nitric oxide synthase (ecNOS); this precedes the production of NO from inducible NOS (iNOS). This leaves a period in early endotoxaemia with a supposed scarcity of NO. The present study was conducted to examine the effects of external supplementation of NO on liver microcirculation and function.
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
|
|
pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
0342-4642
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
24
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1257-63
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:9885877-Animals,
pubmed-meshheading:9885877-Endotoxemia,
pubmed-meshheading:9885877-Infusions, Intravenous,
pubmed-meshheading:9885877-Lipopolysaccharides,
pubmed-meshheading:9885877-Liver Circulation,
pubmed-meshheading:9885877-Liver Diseases,
pubmed-meshheading:9885877-Liver Function Tests,
pubmed-meshheading:9885877-Male,
pubmed-meshheading:9885877-Microcirculation,
pubmed-meshheading:9885877-Nitric Oxide,
pubmed-meshheading:9885877-Nitric Oxide Synthase,
pubmed-meshheading:9885877-Nitroprusside,
pubmed-meshheading:9885877-Rats,
pubmed-meshheading:9885877-Rats, Sprague-Dawley,
pubmed-meshheading:9885877-Vasodilator Agents
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pubmed:year |
1998
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pubmed:articleTitle |
The nitric oxide donor sodium nitroprusside protects against hepatic microcirculatory dysfunction in early endotoxaemia.
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pubmed:affiliation |
Institute for Surgical Research, Rikshospitalet, University of Oslo, Norway.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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