| pubmed-article:9876327 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9876327 | lifeskim:mentions | umls-concept:C0087111 | lld:lifeskim |
| pubmed-article:9876327 | lifeskim:mentions | umls-concept:C0037083 | lld:lifeskim |
| pubmed-article:9876327 | lifeskim:mentions | umls-concept:C0054836 | lld:lifeskim |
| pubmed-article:9876327 | lifeskim:mentions | umls-concept:C1383860 | lld:lifeskim |
| pubmed-article:9876327 | lifeskim:mentions | umls-concept:C0857121 | lld:lifeskim |
| pubmed-article:9876327 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:9876327 | pubmed:dateCreated | 1999-2-2 | lld:pubmed |
| pubmed-article:9876327 | pubmed:abstractText | Treatment with the beta-blocker carvedilol leads to an improvement of outcome and ejection fraction in heart failure. These effects occur without affecting the number of beta-adrenergic receptors, as determined in right ventricular biopsies from patients with heart failure. This study was aimed at investigating the effects of carvedilol on beta-adrenergic signal transduction alterations in a model of left ventricular pressure overload, which is characterized by sympathetic activation and a desensitized beta-adrenergic signal transduction. | lld:pubmed |
| pubmed-article:9876327 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9876327 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9876327 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9876327 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9876327 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:9876327 | pubmed:issn | 0008-6363 | lld:pubmed |
| pubmed-article:9876327 | pubmed:author | pubmed-author:PaulMM | lld:pubmed |
| pubmed-article:9876327 | pubmed:author | pubmed-author:BöhmMM | lld:pubmed |
| pubmed-article:9876327 | pubmed:author | pubmed-author:KnorrAA | lld:pubmed |
| pubmed-article:9876327 | pubmed:author | pubmed-author:ZoliII | lld:pubmed |
| pubmed-article:9876327 | pubmed:author | pubmed-author:FleschMM | lld:pubmed |
| pubmed-article:9876327 | pubmed:author | pubmed-author:van GilstW... | lld:pubmed |
| pubmed-article:9876327 | pubmed:author | pubmed-author:PintoY MYM | lld:pubmed |
| pubmed-article:9876327 | pubmed:author | pubmed-author:MaackCC | lld:pubmed |
| pubmed-article:9876327 | pubmed:author | pubmed-author:TeismanA CAC | lld:pubmed |
| pubmed-article:9876327 | pubmed:author | pubmed-author:EttelbrückSS | lld:pubmed |
| pubmed-article:9876327 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9876327 | pubmed:volume | 40 | lld:pubmed |
| pubmed-article:9876327 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9876327 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9876327 | pubmed:pagination | 146-55 | lld:pubmed |
| pubmed-article:9876327 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:9876327 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9876327 | pubmed:articleTitle | Beta-adrenergic signal transduction following carvedilol treatment in hypertensive cardiac hypertrophy. | lld:pubmed |
| pubmed-article:9876327 | pubmed:affiliation | Klinik III für Innere Medizin der Universität zu Köln, Germany. Michael.Boehm@Medizin.Uni-Koeln.De | lld:pubmed |
| pubmed-article:9876327 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9876327 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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