Switch to
Predicate | Object |
---|---|
rdf:type | |
lifeskim:mentions | |
pubmed:issue |
19 Suppl
|
pubmed:dateCreated |
1999-1-8
|
pubmed:abstractText |
Hypothermic cardioplegia provides myocellular protection, yet postischemic dysfunction remains a significant problem. Interestingly, the subcellular changes in hibernation parallel the altered biology of induced cardiac ischemia but are well tolerated by hibernated mammalian myocardium. An uncharacterized factor derived from hibernating animals, hibernation induction trigger (HIT), has been shown to induce hibernation in active animals and afford myocardial protection after ischemia-reperfusion injury. Therefore, it was of interest to further characterize the cardioprotective effects of HIT in the setting of ischemia-reperfusion injury.
|
pubmed:language |
eng
|
pubmed:journal | |
pubmed:citationSubset |
AIM
|
pubmed:status |
MEDLINE
|
pubmed:month |
Nov
|
pubmed:issn |
0009-7322
|
pubmed:author | |
pubmed:issnType |
Print
|
pubmed:day |
10
|
pubmed:volume |
98
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
II220-3; discussion II223-4
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:9852906-Animals,
pubmed-meshheading:9852906-Female,
pubmed-meshheading:9852906-Heart,
pubmed-meshheading:9852906-Hibernation,
pubmed-meshheading:9852906-Male,
pubmed-meshheading:9852906-Myocardial Ischemia,
pubmed-meshheading:9852906-Myocardial Reperfusion Injury,
pubmed-meshheading:9852906-Rabbits,
pubmed-meshheading:9852906-Time Factors,
pubmed-meshheading:9852906-Ursidae
|
pubmed:year |
1998
|
pubmed:articleTitle |
Hibernation triggers and myocardial protection.
|
pubmed:affiliation |
Section of Thoracic Surgery, University of Michigan Medical Center, Ann Arbor, USA. sbolling@umich.edu
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|