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pubmed-article:9849704pubmed:abstractTextA major pathophysiologic change of pre-eclampsia has been attributed to the overproduction of thromboxane A2 (TXA2) mainly from activated platelets. On the other hand, increased biosynthesis of TXA2 has also been reported from preeclamptic placentas. The systemic role of these different sources of TXA2 has not been clarified. The purpose of this study is to define the changes of TXA2 and the antagonizing prostacyclin (PC) in maternal and fetal circulations.lld:pubmed
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pubmed-article:9849704pubmed:authorpubmed-author:ChangY KYKlld:pubmed
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pubmed-article:9849704pubmed:articleTitleThromboxane and prostacyclin in maternal and fetal circulation in pre-eclampsia.lld:pubmed
pubmed-article:9849704pubmed:affiliationDepartment of Obstetrics and Gynecology, Tri-Service General Hospital, Taipei, Taiwan.lld:pubmed
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pubmed-article:9849704pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed
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