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Predicate | Object |
---|---|
rdf:type | |
lifeskim:mentions | |
pubmed:issue |
1
|
pubmed:dateCreated |
1999-2-17
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pubmed:abstractText |
A major pathophysiologic change of pre-eclampsia has been attributed to the overproduction of thromboxane A2 (TXA2) mainly from activated platelets. On the other hand, increased biosynthesis of TXA2 has also been reported from preeclamptic placentas. The systemic role of these different sources of TXA2 has not been clarified. The purpose of this study is to define the changes of TXA2 and the antagonizing prostacyclin (PC) in maternal and fetal circulations.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Oct
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pubmed:issn |
0020-7292
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
63
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1-6
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:9849704-Adult,
pubmed-meshheading:9849704-Biological Markers,
pubmed-meshheading:9849704-Female,
pubmed-meshheading:9849704-Fetal Blood,
pubmed-meshheading:9849704-Humans,
pubmed-meshheading:9849704-Immunohistochemistry,
pubmed-meshheading:9849704-Maternal-Fetal Exchange,
pubmed-meshheading:9849704-Pre-Eclampsia,
pubmed-meshheading:9849704-Pregnancy,
pubmed-meshheading:9849704-Prostaglandins F,
pubmed-meshheading:9849704-Reference Values,
pubmed-meshheading:9849704-Sensitivity and Specificity,
pubmed-meshheading:9849704-Thromboxane A2
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pubmed:year |
1998
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pubmed:articleTitle |
Thromboxane and prostacyclin in maternal and fetal circulation in pre-eclampsia.
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pubmed:affiliation |
Department of Obstetrics and Gynecology, Tri-Service General Hospital, Taipei, Taiwan.
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pubmed:publicationType |
Journal Article,
Clinical Trial,
Controlled Clinical Trial,
Research Support, Non-U.S. Gov't
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