| pubmed-article:9781002 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9781002 | lifeskim:mentions | umls-concept:C0019080 | lld:lifeskim |
| pubmed-article:9781002 | lifeskim:mentions | umls-concept:C0225336 | lld:lifeskim |
| pubmed-article:9781002 | lifeskim:mentions | umls-concept:C0699819 | lld:lifeskim |
| pubmed-article:9781002 | lifeskim:mentions | umls-concept:C0009498 | lld:lifeskim |
| pubmed-article:9781002 | lifeskim:mentions | umls-concept:C0022116 | lld:lifeskim |
| pubmed-article:9781002 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
| pubmed-article:9781002 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:9781002 | lifeskim:mentions | umls-concept:C0035273 | lld:lifeskim |
| pubmed-article:9781002 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:9781002 | lifeskim:mentions | umls-concept:C1292733 | lld:lifeskim |
| pubmed-article:9781002 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:9781002 | pubmed:dateCreated | 1998-11-5 | lld:pubmed |
| pubmed-article:9781002 | pubmed:abstractText | Complement, a nonspecific immune response, is activated during hemorrhage/resuscitation (HEM/RES) and is involved in cellular damage. We hypothesized that activated complement injures endothelial cells (ETCs) and is responsible for intestinal microvascular hypoperfusion after HEM/RES. | lld:pubmed |
| pubmed-article:9781002 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9781002 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9781002 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:9781002 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9781002 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:9781002 | pubmed:issn | 0039-6060 | lld:pubmed |
| pubmed-article:9781002 | pubmed:author | pubmed-author:HarrisP DPD | lld:pubmed |
| pubmed-article:9781002 | pubmed:author | pubmed-author:GarrisonR NRN | lld:pubmed |
| pubmed-article:9781002 | pubmed:author | pubmed-author:WilsonM AMA | lld:pubmed |
| pubmed-article:9781002 | pubmed:author | pubmed-author:SpainD ADA | lld:pubmed |
| pubmed-article:9781002 | pubmed:author | pubmed-author:FruchtermanT... | lld:pubmed |
| pubmed-article:9781002 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9781002 | pubmed:volume | 124 | lld:pubmed |
| pubmed-article:9781002 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9781002 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9781002 | pubmed:pagination | 782-91; discussion 791-2 | lld:pubmed |
| pubmed-article:9781002 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:9781002 | pubmed:meshHeading | pubmed-meshheading:9781002-... | lld:pubmed |
| pubmed-article:9781002 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9781002 | pubmed:articleTitle | Complement inhibition prevents gut ischemia and endothelial cell dysfunction after hemorrhage/resuscitation. | lld:pubmed |
| pubmed-article:9781002 | pubmed:affiliation | Department of Surgery, University of Louisville, KY 40292, USA. | lld:pubmed |
| pubmed-article:9781002 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9781002 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:9781002 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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