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rdf:type |
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lifeskim:mentions |
|
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pubmed:issue |
4
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pubmed:dateCreated |
1998-11-5
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pubmed:abstractText |
Complement, a nonspecific immune response, is activated during hemorrhage/resuscitation (HEM/RES) and is involved in cellular damage. We hypothesized that activated complement injures endothelial cells (ETCs) and is responsible for intestinal microvascular hypoperfusion after HEM/RES.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
|
|
pubmed:status |
MEDLINE
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pubmed:month |
Oct
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pubmed:issn |
0039-6060
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pubmed:author |
|
|
pubmed:issnType |
Print
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pubmed:volume |
124
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pubmed:owner |
NLM
|
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pubmed:authorsComplete |
Y
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pubmed:pagination |
782-91; discussion 791-2
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:9781002-Acetylcholine,
pubmed-meshheading:9781002-Animals,
pubmed-meshheading:9781002-Complement Activation,
pubmed-meshheading:9781002-Endothelium, Vascular,
pubmed-meshheading:9781002-Intestine, Small,
pubmed-meshheading:9781002-Ischemia,
pubmed-meshheading:9781002-Male,
pubmed-meshheading:9781002-Microcirculation,
pubmed-meshheading:9781002-Nitroprusside,
pubmed-meshheading:9781002-Rats,
pubmed-meshheading:9781002-Rats, Sprague-Dawley,
pubmed-meshheading:9781002-Receptors, Complement,
pubmed-meshheading:9781002-Resuscitation,
pubmed-meshheading:9781002-Shock, Hemorrhagic,
pubmed-meshheading:9781002-Vasodilation,
pubmed-meshheading:9781002-Vasodilator Agents
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pubmed:year |
1998
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pubmed:articleTitle |
Complement inhibition prevents gut ischemia and endothelial cell dysfunction after hemorrhage/resuscitation.
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pubmed:affiliation |
Department of Surgery, University of Louisville, KY 40292, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, Non-P.H.S.,
Research Support, Non-U.S. Gov't
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