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rdf:type |
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lifeskim:mentions |
umls-concept:C0017262,
umls-concept:C0017337,
umls-concept:C0019169,
umls-concept:C0035696,
umls-concept:C0086418,
umls-concept:C0185117,
umls-concept:C0205374,
umls-concept:C0227525,
umls-concept:C0441889,
umls-concept:C1514248,
umls-concept:C2911684
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pubmed:issue |
7
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pubmed:dateCreated |
1998-11-17
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pubmed:abstractText |
Hepatitis B virus (HBV) infection is closely related to the development of not only acute or chronic hepatitis, but also hepatocellular carcinoma. Among the HBV genes, the X gene has been implicated in the carcinogenicity of this virus as a major causative factor by its ability to activate viral and cellular genes in trans via protein-protein interaction with cellular factors without binding to DNA.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Antigens, Polyomavirus Transforming,
http://linkedlifedata.com/resource/pubmed/chemical/Chloramphenicol O-Acetyltransferase,
http://linkedlifedata.com/resource/pubmed/chemical/Dactinomycin,
http://linkedlifedata.com/resource/pubmed/chemical/Nucleic Acid Synthesis Inhibitors,
http://linkedlifedata.com/resource/pubmed/chemical/Peptide Elongation Factor 1,
http://linkedlifedata.com/resource/pubmed/chemical/Peptide Elongation Factors,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Viral,
http://linkedlifedata.com/resource/pubmed/chemical/Trans-Activators,
http://linkedlifedata.com/resource/pubmed/chemical/hepatitis B virus X protein
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pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
1356-9597
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
3
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
477-84
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pubmed:dateRevised |
2008-11-21
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pubmed:meshHeading |
pubmed-meshheading:9753428-Antigens, Polyomavirus Transforming,
pubmed-meshheading:9753428-Carcinoma, Hepatocellular,
pubmed-meshheading:9753428-Chloramphenicol O-Acetyltransferase,
pubmed-meshheading:9753428-Dactinomycin,
pubmed-meshheading:9753428-Gene Expression Regulation, Viral,
pubmed-meshheading:9753428-Genes, Viral,
pubmed-meshheading:9753428-Hepatitis B virus,
pubmed-meshheading:9753428-Hepatoblastoma,
pubmed-meshheading:9753428-Humans,
pubmed-meshheading:9753428-Liver,
pubmed-meshheading:9753428-Nucleic Acid Synthesis Inhibitors,
pubmed-meshheading:9753428-Peptide Elongation Factor 1,
pubmed-meshheading:9753428-Peptide Elongation Factors,
pubmed-meshheading:9753428-Promoter Regions, Genetic,
pubmed-meshheading:9753428-RNA, Messenger,
pubmed-meshheading:9753428-RNA, Viral,
pubmed-meshheading:9753428-Sequence Deletion,
pubmed-meshheading:9753428-Simian virus 40,
pubmed-meshheading:9753428-Trans-Activators,
pubmed-meshheading:9753428-Transcription, Genetic,
pubmed-meshheading:9753428-Transfection,
pubmed-meshheading:9753428-Tumor Cells, Cultured
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pubmed:year |
1998
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pubmed:articleTitle |
Post-transcriptional control of the level of mRNA by hepatitis B virus X gene in the transient expression system using human hepatic cells.
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pubmed:affiliation |
Department of Gene Research, The Cancer Institute, JFCR, Tokyo, Japan.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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