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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
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pubmed:dateCreated |
1998-11-17
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pubmed:abstractText |
Chronic renal allograft rejection is characterized histologically by transplantation-associated arteriosclerosis and glomerulosclerosis (Tx-AA and Tx-AGS). Recent studies in animal models implicate the mitogenic and pressor actions of endothelin-1 (ET-1) in Tx-AA. In humans, however, a link between elevated ET-1 secretion and Tx-AA or Tx-AGS remains unclear. In this study we analyzed expression of ET-1 in the vasculature of renal transplant patients with chronic or acute rejection and in normal controls.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Sep
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pubmed:issn |
0085-2538
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
54
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
960-71
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:9734624-Adolescent,
pubmed-meshheading:9734624-Adult,
pubmed-meshheading:9734624-Aged,
pubmed-meshheading:9734624-Arteriosclerosis,
pubmed-meshheading:9734624-Cells, Cultured,
pubmed-meshheading:9734624-Cytokines,
pubmed-meshheading:9734624-Endothelin-1,
pubmed-meshheading:9734624-Female,
pubmed-meshheading:9734624-Glomerular Mesangium,
pubmed-meshheading:9734624-Graft Rejection,
pubmed-meshheading:9734624-Humans,
pubmed-meshheading:9734624-Kidney Transplantation,
pubmed-meshheading:9734624-Male,
pubmed-meshheading:9734624-Middle Aged,
pubmed-meshheading:9734624-Muscle, Smooth, Vascular,
pubmed-meshheading:9734624-Transplantation, Homologous
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pubmed:year |
1998
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pubmed:articleTitle |
Elevated neointimal endothelin-1 in transplantation-associated arteriosclerosis of renal allograft recipients.
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pubmed:affiliation |
Department of Medicine, School of Medicine, Case Western Reserve University, and University Hospitals of Cleveland, Ohio 44106, USA. mss5@po.cwru.edu
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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