| pubmed-article:9716532 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9716532 | lifeskim:mentions | umls-concept:C0017347 | lld:lifeskim |
| pubmed-article:9716532 | lifeskim:mentions | umls-concept:C0105770 | lld:lifeskim |
| pubmed-article:9716532 | lifeskim:mentions | umls-concept:C1160214 | lld:lifeskim |
| pubmed-article:9716532 | lifeskim:mentions | umls-concept:C0162610 | lld:lifeskim |
| pubmed-article:9716532 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:9716532 | lifeskim:mentions | umls-concept:C1334043 | lld:lifeskim |
| pubmed-article:9716532 | pubmed:issue | 18 | lld:pubmed |
| pubmed-article:9716532 | pubmed:dateCreated | 1998-10-30 | lld:pubmed |
| pubmed-article:9716532 | pubmed:abstractText | In C. elegans, the epithelial Pn.p cells adopt either a vulval precursor cell fate or fuse with the surrounding hypodermis (the F fate). Our results suggest that a Wnt signal transduced through a pathway involving the beta-catenin homolog BAR-1 controls whether P3.p through P8.p adopt the vulval precursor cell fate. In bar-1 mutants, P3.p through P8.p can adopt F fates instead of vulval precursor cell fates. The Wnt/bar-1 signaling pathway acts by regulating the expression of the Hox gene lin-39, since bar-1 is required for LIN-39 expression and forced lin-39 expression rescues the bar-1 mutant phenotype. LIN-39 activity is also regulated by the anchor cell signal/let-23 receptor tyrosine kinase/let-60 Ras signaling pathway. Our genetic and molecular experiments show that the vulval precursor cells can integrate the input from the BAR-1 and LET-60 Ras signaling pathways by coordinately regulating activity of the common target LIN-39 Hox. | lld:pubmed |
| pubmed-article:9716532 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9716532 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:9716532 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9716532 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9716532 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:9716532 | pubmed:issn | 0950-1991 | lld:pubmed |
| pubmed-article:9716532 | pubmed:author | pubmed-author:KimS KSK | lld:pubmed |
| pubmed-article:9716532 | pubmed:author | pubmed-author:KenyonCC | lld:pubmed |
| pubmed-article:9716532 | pubmed:author | pubmed-author:EisenmannD... | lld:pubmed |
| pubmed-article:9716532 | pubmed:author | pubmed-author:SimskeJ SJS | lld:pubmed |
| pubmed-article:9716532 | pubmed:author | pubmed-author:MaloofJ NJN | lld:pubmed |
| pubmed-article:9716532 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9716532 | pubmed:volume | 125 | lld:pubmed |
| pubmed-article:9716532 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9716532 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9716532 | pubmed:pagination | 3667-80 | lld:pubmed |
| pubmed-article:9716532 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:9716532 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9716532 | pubmed:articleTitle | The beta-catenin homolog BAR-1 and LET-60 Ras coordinately regulate the Hox gene lin-39 during Caenorhabditis elegans vulval development. | lld:pubmed |
| pubmed-article:9716532 | pubmed:affiliation | Department of Developmental Biology, Stanford University, Stanford, CA 94305, USA. | lld:pubmed |
| pubmed-article:9716532 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9716532 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:9716532 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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