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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
1998-8-26
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pubmed:abstractText |
Numerous studies support the concept that cardiac angiotensin-converting enzyme (ACE) is involved in the pathophysiology of left ventricular hypertrophy. However, the pulmonary vasculature is considered to be the most prominent site of ACE expression. We thus examined the tissue specificity of ACE regulation in rats with severe cardiac pressure overload hypertrophy in transition to cardiac failure with secondary pulmonary hypertension.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0008-6363
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
38
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
125-32
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:9683914-Animals,
pubmed-meshheading:9683914-Blotting, Northern,
pubmed-meshheading:9683914-Body Weight,
pubmed-meshheading:9683914-Diuretics,
pubmed-meshheading:9683914-Furosemide,
pubmed-meshheading:9683914-Hypertension, Pulmonary,
pubmed-meshheading:9683914-Hypertrophy, Left Ventricular,
pubmed-meshheading:9683914-Lung,
pubmed-meshheading:9683914-Male,
pubmed-meshheading:9683914-Myocardium,
pubmed-meshheading:9683914-Organ Size,
pubmed-meshheading:9683914-Peptidyl-Dipeptidase A,
pubmed-meshheading:9683914-Polymerase Chain Reaction,
pubmed-meshheading:9683914-Pulmonary Wedge Pressure,
pubmed-meshheading:9683914-RNA, Messenger,
pubmed-meshheading:9683914-Rats,
pubmed-meshheading:9683914-Rats, Wistar,
pubmed-meshheading:9683914-Renin
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pubmed:year |
1998
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pubmed:articleTitle |
Reciprocal regulation of pulmonary and cardiac angiotensin-converting enzyme in rats with severe left ventricular hypertrophy.
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pubmed:affiliation |
Klinik und Poliklinik für Innere Medizin II, Klinikum der Universität, Regensburg, Germany.
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pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't
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