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rdf:type |
|
|
lifeskim:mentions |
|
|
pubmed:issue |
25
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pubmed:dateCreated |
1998-7-15
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|
pubmed:abstractText |
As endothelin binds to ET(A) receptors, it stimulates vascular smooth muscle cell proliferation and may thus be pivotally involved in the pathogenesis of restenosis. This study assessed the ability of a potent and selective ET(A) antagonist to reduce neointimal hyperplasia in a porcine coronary artery stented injury model.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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|
pubmed:month |
Jun
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pubmed:issn |
0009-7322
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|
pubmed:author |
pubmed-author:BurkaS ASA,
pubmed-author:CamrudA RAR,
pubmed-author:CamrudL JLJ,
pubmed-author:CarlsonP JPJ,
pubmed-author:HolmesD RDRJr,
pubmed-author:JohnsonJJ,
pubmed-author:LermanAA,
pubmed-author:McKennaC JCJ,
pubmed-author:OpgenorthT JTJ,
pubmed-author:PadleyR JRJ,
pubmed-author:SchwartzR SRS
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pubmed:issnType |
Print
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|
pubmed:day |
30
|
|
pubmed:volume |
97
|
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pubmed:owner |
NLM
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|
pubmed:authorsComplete |
Y
|
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pubmed:pagination |
2551-6
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pubmed:dateRevised |
2003-11-14
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pubmed:meshHeading |
pubmed-meshheading:9657476-Administration, Oral,
pubmed-meshheading:9657476-Animals,
pubmed-meshheading:9657476-Blood Pressure,
pubmed-meshheading:9657476-Coronary Disease,
pubmed-meshheading:9657476-Coronary Vessels,
pubmed-meshheading:9657476-Disease Models, Animal,
pubmed-meshheading:9657476-Hyperplasia,
pubmed-meshheading:9657476-Random Allocation,
pubmed-meshheading:9657476-Receptors, Endothelin,
pubmed-meshheading:9657476-Regression Analysis,
pubmed-meshheading:9657476-Stents,
pubmed-meshheading:9657476-Swine,
pubmed-meshheading:9657476-Tunica Intima
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pubmed:year |
1998
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pubmed:articleTitle |
Selective ET(A) receptor antagonism reduces neointimal hyperplasia in a porcine coronary stent model.
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pubmed:affiliation |
Division of Cardiovascular Diseases, Mayo Clinic and Mayo Foundation, Rochester, Minn, USA.
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pubmed:publicationType |
Journal Article
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