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| pubmed-article:9648076 | lifeskim:mentions | umls-concept:C0599635 | lld:lifeskim |
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| pubmed-article:9648076 | lifeskim:mentions | umls-concept:C0597484 | lld:lifeskim |
| pubmed-article:9648076 | lifeskim:mentions | umls-concept:C0205349 | lld:lifeskim |
| pubmed-article:9648076 | lifeskim:mentions | umls-concept:C0205385 | lld:lifeskim |
| pubmed-article:9648076 | lifeskim:mentions | umls-concept:C1517004 | lld:lifeskim |
| pubmed-article:9648076 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
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| pubmed-article:9648076 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:9648076 | pubmed:dateCreated | 1998-9-16 | lld:pubmed |
| pubmed-article:9648076 | pubmed:abstractText | Several pathophysiological states associated with deranged water balance are associated with altered expression and/or intracellular distribution of aquaporin water channels. The possible role of dysregulation of thick ascending limb NaCl transporters, which are responsible for countercurrent multiplication in the kidney, has not been evaluated. | lld:pubmed |
| pubmed-article:9648076 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:9648076 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9648076 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9648076 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:9648076 | pubmed:issn | 0085-2538 | lld:pubmed |
| pubmed-article:9648076 | pubmed:author | pubmed-author:NielsenSS | lld:pubmed |
| pubmed-article:9648076 | pubmed:author | pubmed-author:AndrewsPP | lld:pubmed |
| pubmed-article:9648076 | pubmed:author | pubmed-author:KnepperMM | lld:pubmed |
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| pubmed-article:9648076 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9648076 | pubmed:volume | 54 | lld:pubmed |
| pubmed-article:9648076 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9648076 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9648076 | pubmed:pagination | 170-9 | lld:pubmed |
| pubmed-article:9648076 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:9648076 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9648076 | pubmed:articleTitle | Concentrating defect in experimental nephrotic syndrone: altered expression of aquaporins and thick ascending limb Na+ transporters. | lld:pubmed |
| pubmed-article:9648076 | pubmed:affiliation | Laboratory of Kidney and Electrolyte Metabolism, National Heart, Lung and Blood Institute, National Institute of Health, Bethesda, Maryland, USA. | lld:pubmed |
| pubmed-article:9648076 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9648076 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:9648076 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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