| pubmed-article:9632395 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9632395 | lifeskim:mentions | umls-concept:C0021756 | lld:lifeskim |
| pubmed-article:9632395 | lifeskim:mentions | umls-concept:C0162508 | lld:lifeskim |
| pubmed-article:9632395 | lifeskim:mentions | umls-concept:C0035696 | lld:lifeskim |
| pubmed-article:9632395 | lifeskim:mentions | umls-concept:C0031727 | lld:lifeskim |
| pubmed-article:9632395 | lifeskim:mentions | umls-concept:C1704259 | lld:lifeskim |
| pubmed-article:9632395 | lifeskim:mentions | umls-concept:C1705987 | lld:lifeskim |
| pubmed-article:9632395 | pubmed:issue | 5371 | lld:pubmed |
| pubmed-article:9632395 | pubmed:dateCreated | 1998-7-15 | lld:pubmed |
| pubmed-article:9632395 | pubmed:abstractText | Signaling pathways that stabilize interleukin-2 (IL-2) messenger RNA (mRNA) in activated T cells were examined. IL-2 mRNA contains at least two cis elements that mediated its stabilization in response to different signals, including activation of c-Jun amino-terminal kinase (JNK). This response was mediated through a cis element encompassing the 5' untranslated region (UTR) and the beginning of the coding region. IL-2 transcripts lacking this 5' element no longer responded to JNK activation but were still responsive to other signals generated during T cell activation, which were probably sensed through the 3' UTR. Thus, multiple elements within IL-2 mRNA modulate its stability in a combinatorial manner, and the JNK pathway controls turnover as well as synthesis of IL-2 mRNA. | lld:pubmed |
| pubmed-article:9632395 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:9632395 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9632395 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:9632395 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:9632395 | pubmed:author | pubmed-author:ChenC YCY | lld:pubmed |
| pubmed-article:9632395 | pubmed:author | pubmed-author:KarinMM | lld:pubmed |
| pubmed-article:9632395 | pubmed:author | pubmed-author:WuZZ | lld:pubmed |
| pubmed-article:9632395 | pubmed:author | pubmed-author:Del... | lld:pubmed |
| pubmed-article:9632395 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9632395 | pubmed:day | 19 | lld:pubmed |
| pubmed-article:9632395 | pubmed:volume | 280 | lld:pubmed |
| pubmed-article:9632395 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9632395 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9632395 | pubmed:pagination | 1945-9 | lld:pubmed |
| pubmed-article:9632395 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:9632395 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9632395 | pubmed:articleTitle | Stabilization of interleukin-2 mRNA by the c-Jun NH2-terminal kinase pathway. | lld:pubmed |
| pubmed-article:9632395 | pubmed:affiliation | Department of Pharmacology, School of Medicine, University of California, San Diego, La Jolla, CA 92093, USA. | lld:pubmed |
| pubmed-article:9632395 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9632395 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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