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| pubmed-article:9577951 | lifeskim:mentions | umls-concept:C0175677 | lld:lifeskim |
| pubmed-article:9577951 | lifeskim:mentions | umls-concept:C0001675 | lld:lifeskim |
| pubmed-article:9577951 | lifeskim:mentions | umls-concept:C0024660 | lld:lifeskim |
| pubmed-article:9577951 | lifeskim:mentions | umls-concept:C0225828 | lld:lifeskim |
| pubmed-article:9577951 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
| pubmed-article:9577951 | lifeskim:mentions | umls-concept:C0242184 | lld:lifeskim |
| pubmed-article:9577951 | lifeskim:mentions | umls-concept:C0683598 | lld:lifeskim |
| pubmed-article:9577951 | pubmed:issue | 14 | lld:pubmed |
| pubmed-article:9577951 | pubmed:dateCreated | 1998-5-27 | lld:pubmed |
| pubmed-article:9577951 | pubmed:abstractText | Previous studies in isolated cardiac myocytes have shown that tumor necrosis factor (TNF)-alpha provokes increased expression of 27- and 70-kD stress proteins as well as manganese superoxide dismutase, suggesting that TNF-alpha might play a role in mediating stress responses in the heart. | lld:pubmed |
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| pubmed-article:9577951 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:9577951 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:9577951 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9577951 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:9577951 | pubmed:issn | 0009-7322 | lld:pubmed |
| pubmed-article:9577951 | pubmed:author | pubmed-author:NakanoMM | lld:pubmed |
| pubmed-article:9577951 | pubmed:author | pubmed-author:PILZCC | lld:pubmed |
| pubmed-article:9577951 | pubmed:author | pubmed-author:KnowltonA AAA | lld:pubmed |
| pubmed-article:9577951 | pubmed:author | pubmed-author:DibbsZZ | lld:pubmed |
| pubmed-article:9577951 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9577951 | pubmed:day | 14 | lld:pubmed |
| pubmed-article:9577951 | pubmed:volume | 97 | lld:pubmed |
| pubmed-article:9577951 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9577951 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9577951 | pubmed:pagination | 1392-400 | lld:pubmed |
| pubmed-article:9577951 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:9577951 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9577951 | pubmed:articleTitle | Tumor necrosis factor-alpha confers resistance to hypoxic injury in the adult mammalian cardiac myocyte. | lld:pubmed |
| pubmed-article:9577951 | pubmed:affiliation | Department of Medicine, Veterans Administration Medical Center, Baylor College of Medicine, Houston, Tex 77030, USA. | lld:pubmed |
| pubmed-article:9577951 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9577951 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:9577951 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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