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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
13
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pubmed:dateCreated |
1998-5-13
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pubmed:abstractText |
Loss of endothelial NO production after arterial injury may contribute to restenosis, characterized by neointima formation and elastic recoil. Adenovirus-mediated transfer of the gene encoding NO synthase (NOS) in balloon-injured arteries may restore NO production and inhibit neointima formation.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0009-7322
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:day |
7
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pubmed:volume |
97
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1274-81
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:9570198-Adenoviridae,
pubmed-meshheading:9570198-Angioplasty, Balloon,
pubmed-meshheading:9570198-Animals,
pubmed-meshheading:9570198-Aorta,
pubmed-meshheading:9570198-Cell Division,
pubmed-meshheading:9570198-Endothelium, Vascular,
pubmed-meshheading:9570198-Gene Transfer Techniques,
pubmed-meshheading:9570198-Humans,
pubmed-meshheading:9570198-Muscle, Smooth, Vascular,
pubmed-meshheading:9570198-Nitric Oxide Synthase,
pubmed-meshheading:9570198-Nitric Oxide Synthase Type III,
pubmed-meshheading:9570198-Rats,
pubmed-meshheading:9570198-Tunica Intima
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pubmed:year |
1998
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pubmed:articleTitle |
Human endothelial nitric oxide synthase gene transfer inhibits vascular smooth muscle cell proliferation and neointima formation after balloon injury in rats.
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pubmed:affiliation |
Cardiac Unit, University Hospital Gasthuisberg, University of Leuven, Belgium. stefan.janssens@med.kuleuven.ac.be
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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