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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
1998-5-14
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pubmed:abstractText |
This study was undertaken to determine whether suppression of complement activation with soluble human complement receptor type I reduces myocardial damage during the revascularization of ischemic myocardium.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0003-4975
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
65
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
973-7
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:9564912-Acidosis,
pubmed-meshheading:9564912-Animals,
pubmed-meshheading:9564912-Cardiomyopathies,
pubmed-meshheading:9564912-Complement Activation,
pubmed-meshheading:9564912-Complement Inactivator Proteins,
pubmed-meshheading:9564912-Coronary Disease,
pubmed-meshheading:9564912-Heart Arrest, Induced,
pubmed-meshheading:9564912-Hemolysis,
pubmed-meshheading:9564912-Humans,
pubmed-meshheading:9564912-Hydrogen-Ion Concentration,
pubmed-meshheading:9564912-Infusions, Intravenous,
pubmed-meshheading:9564912-Myocardial Contraction,
pubmed-meshheading:9564912-Myocardial Ischemia,
pubmed-meshheading:9564912-Myocardial Reperfusion,
pubmed-meshheading:9564912-Myocardial Reperfusion Injury,
pubmed-meshheading:9564912-Myocardium,
pubmed-meshheading:9564912-Necrosis,
pubmed-meshheading:9564912-Random Allocation,
pubmed-meshheading:9564912-Receptors, Complement,
pubmed-meshheading:9564912-Swine
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pubmed:year |
1998
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pubmed:articleTitle |
Soluble complement receptor type I limits damage during revascularization of ischemic myocardium.
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pubmed:affiliation |
Department of Cardiothoracic Surgery, Boston Medical Center, Massachusetts 02118, USA.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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