Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
8
pubmed:dateCreated
1998-5-13
pubmed:abstractText
We identified a set of cytokinin-insensitive mutants by using a screen based on the ethylene-mediated triple response observed after treatment with low levels of cytokinins. One group of these mutants disrupts ACS5, a member of the Arabidopsis gene family that encodes 1-aminocyclopropane-1-carboxylate synthase, the first enzyme in ethylene biosynthesis. The ACS5 isoform is mainly responsible for the sustained rise in ethylene biosynthesis observed in response to low levels of cytokinin and appears to be regulated primarily by a posttranscriptional mechanism. Furthermore, the dominant ethylene-overproducing mutant eto2 was found to be the result of an alteration of the carboxy terminus of ACS5, suggesting that this domain acts as a negative regulator of ACS5 function.
pubmed:commentsCorrections
http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-11541139, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-1357670, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-1421146, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-1438312, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-1549612, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-16667643, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-1711229, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-1762159, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-1990006, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-2030957, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-2152173, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-2191304, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-3327686, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-7516081, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-7642574, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-7765118, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-7770519, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-7858201, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-7858205, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-8120053, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-8188214, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-8211181, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-8292780, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-8366090, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-8431946, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-8525372, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-8566772, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-8624515, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-8628251, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-9011084, http://linkedlifedata.com/resource/pubmed/commentcorrection/9539813-9215635
pubmed:keyword
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Apr
pubmed:issn
0027-8424
pubmed:author
pubmed:issnType
Print
pubmed:day
14
pubmed:volume
95
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
4766-71
pubmed:dateRevised
2010-9-13
pubmed:meshHeading
pubmed:year
1998
pubmed:articleTitle
Recessive and dominant mutations in the ethylene biosynthetic gene ACS5 of Arabidopsis confer cytokinin insensitivity and ethylene overproduction, respectively.
pubmed:affiliation
Department of Biological Sciences, Laboratory for Molecular Biology, University of Illinois, Chicago, IL 60607, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, Non-P.H.S.