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rdf:type |
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lifeskim:mentions |
umls-concept:C0017262,
umls-concept:C0036945,
umls-concept:C0042382,
umls-concept:C0078058,
umls-concept:C0185117,
umls-concept:C0205217,
umls-concept:C0215848,
umls-concept:C0332281,
umls-concept:C0521457,
umls-concept:C1171892,
umls-concept:C1383860,
umls-concept:C1522564,
umls-concept:C2911684
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pubmed:issue |
3
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pubmed:dateCreated |
1998-4-22
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pubmed:abstractText |
Our purpose was to determine whether the increase in fetal cardiac mass and cardiac output in chronic anemia is accompanied by changes in capillary density or size or changes in levels of vascular endothelial growth factor and hypoxia-inducible factor 1, a basic helix-loop-helix transcription factor that has previously been shown to activate vascular endothelial growth factor gene transcription when cultured cells are subjected to hypoxia.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/DNA-Binding Proteins,
http://linkedlifedata.com/resource/pubmed/chemical/Endothelial Growth Factors,
http://linkedlifedata.com/resource/pubmed/chemical/HIF1A protein, human,
http://linkedlifedata.com/resource/pubmed/chemical/Hypoxia-Inducible Factor 1,
http://linkedlifedata.com/resource/pubmed/chemical/Hypoxia-Inducible Factor 1, alpha...,
http://linkedlifedata.com/resource/pubmed/chemical/Lymphokines,
http://linkedlifedata.com/resource/pubmed/chemical/Nuclear Proteins,
http://linkedlifedata.com/resource/pubmed/chemical/Transcription Factors,
http://linkedlifedata.com/resource/pubmed/chemical/Vascular Endothelial Growth Factor A,
http://linkedlifedata.com/resource/pubmed/chemical/Vascular Endothelial Growth Factors
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pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
0002-9378
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
178
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
527-34
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:9539521-Anemia,
pubmed-meshheading:9539521-Animals,
pubmed-meshheading:9539521-Capillaries,
pubmed-meshheading:9539521-Cardiac Output,
pubmed-meshheading:9539521-Cardiomegaly,
pubmed-meshheading:9539521-Cells, Cultured,
pubmed-meshheading:9539521-Chronic Disease,
pubmed-meshheading:9539521-Coronary Circulation,
pubmed-meshheading:9539521-DNA-Binding Proteins,
pubmed-meshheading:9539521-Endothelial Growth Factors,
pubmed-meshheading:9539521-Female,
pubmed-meshheading:9539521-Fetal Diseases,
pubmed-meshheading:9539521-Helix-Loop-Helix Motifs,
pubmed-meshheading:9539521-Humans,
pubmed-meshheading:9539521-Hypoxia-Inducible Factor 1,
pubmed-meshheading:9539521-Hypoxia-Inducible Factor 1, alpha Subunit,
pubmed-meshheading:9539521-Lymphokines,
pubmed-meshheading:9539521-Myocardium,
pubmed-meshheading:9539521-Nuclear Proteins,
pubmed-meshheading:9539521-Sheep,
pubmed-meshheading:9539521-Transcription Factors,
pubmed-meshheading:9539521-Vascular Endothelial Growth Factor A,
pubmed-meshheading:9539521-Vascular Endothelial Growth Factors
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pubmed:year |
1998
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pubmed:articleTitle |
Cardiac hypertrophy in chronically anemic fetal sheep: Increased vascularization is associated with increased myocardial expression of vascular endothelial growth factor and hypoxia-inducible factor 1.
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pubmed:affiliation |
Department of Obstetrics and Gynecology and the Congenital Heart Center, Oregon Health Sciences University, Portland 97201-3098, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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