rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
3
|
pubmed:dateCreated |
1998-4-16
|
pubmed:abstractText |
The hypothesis was that Na+/H+ exchange occurring during normothermic cardioplegia contributes to the development of myocardial edema during subsequent reperfusion and impairs functional recovery.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Mar
|
pubmed:issn |
0022-5223
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
115
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
709-15
|
pubmed:dateRevised |
2007-11-15
|
pubmed:meshHeading |
pubmed-meshheading:9535460-Animals,
pubmed-meshheading:9535460-Edema, Cardiac,
pubmed-meshheading:9535460-Guanidines,
pubmed-meshheading:9535460-Heart Arrest, Induced,
pubmed-meshheading:9535460-Hemodynamics,
pubmed-meshheading:9535460-L-Lactate Dehydrogenase,
pubmed-meshheading:9535460-Myocardial Reperfusion Injury,
pubmed-meshheading:9535460-Rats,
pubmed-meshheading:9535460-Rats, Sprague-Dawley,
pubmed-meshheading:9535460-Sodium-Hydrogen Antiporter,
pubmed-meshheading:9535460-Sulfones,
pubmed-meshheading:9535460-Ventricular Function, Left
|
pubmed:year |
1998
|
pubmed:articleTitle |
Sodium/hydrogen exchanger inhibition reduces myocardial reperfusion edema after normothermic cardioplegia.
|
pubmed:affiliation |
Laboratorio de Cardiologia Experimental B. Servicio de Cardiologia, Hospital General Universitari Vall d'Hebron, Barcelona, Spain.
|
pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, Non-U.S. Gov't
|