Linked Life Data

9445354

Source:http://linkedlifedata.com/resource/pubmed/id/9445354

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pubmed-article:9445354pubmed:abstractTextArterial hypoxia mediates cerebral arteriolar dilation primarily via mechanisms involving activation of ATP-sensitive K+ channels (K[ATP]), which we have shown to be sensitive to ischemic stress. In this study, we determined whether ischemia/reperfusion alters cerebral arteriolar responses to arterial hypoxia in anesthetized piglets. Since adenosine plays an important role in cerebrovascular responses to hypoxia, we also determined whether adenosine-induced arteriolar dilation is affected by ischemic stress. We tested the hypothesis that reductions in cerebral arteriolar dilator responses after ischemia would be proportional to the contribution of K(ATP) to hypoxia and adenosine.lld:pubmed
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pubmed-article:9445354pubmed:pagination222-7; discussion 227-8lld:pubmed
pubmed-article:9445354pubmed:dateRevised2010-11-18lld:pubmed
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pubmed-article:9445354pubmed:articleTitleEffects of ischemia on cerebral arteriolar dilation to arterial hypoxia in piglets.lld:pubmed
pubmed-article:9445354pubmed:affiliationDepartment of Physiology and Pharmacology, Bowman Gray School of Medicine, Wake Forest University, Winston-Salem, NC 27157-1083, USA.lld:pubmed
pubmed-article:9445354pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:9445354pubmed:publicationTypeResearch Support, U.S. Gov't, P.H.S.lld:pubmed
pubmed-article:9445354pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed
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