rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
5
|
pubmed:dateCreated |
1998-1-2
|
pubmed:abstractText |
Mycobacteria have been considered a possible etiological agent in Crohn's disease. Since cross-reactivity between epitopes of mycobacterial and self-heat shock protein might represent a potential disease mechanism, we determined the cellular and humoral immune responses to the mycobacterial and the human 60-kD heat shock protein, as well as various control antigens.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:issn |
0012-2823
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
58
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
469-75
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:9383639-Adult,
pubmed-meshheading:9383639-Antibody Formation,
pubmed-meshheading:9383639-Antigens, Bacterial,
pubmed-meshheading:9383639-Case-Control Studies,
pubmed-meshheading:9383639-Chaperonin 60,
pubmed-meshheading:9383639-Colitis, Ulcerative,
pubmed-meshheading:9383639-Crohn Disease,
pubmed-meshheading:9383639-Enzyme-Linked Immunosorbent Assay,
pubmed-meshheading:9383639-Humans,
pubmed-meshheading:9383639-Lymphocyte Activation,
pubmed-meshheading:9383639-Lymphocytes,
pubmed-meshheading:9383639-Mycobacterium tuberculosis
|
pubmed:year |
1997
|
pubmed:articleTitle |
Cellular and humoral immunity to the 60-kD heat shock protein in inflammatory bowel disease.
|
pubmed:affiliation |
1st Department of Medicine, Johannes Gutenberg University, Mainz, Germany.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|