| pubmed-article:9350434 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9350434 | lifeskim:mentions | umls-concept:C0079904 | lld:lifeskim |
| pubmed-article:9350434 | lifeskim:mentions | umls-concept:C1704259 | lld:lifeskim |
| pubmed-article:9350434 | lifeskim:mentions | umls-concept:C1160185 | lld:lifeskim |
| pubmed-article:9350434 | lifeskim:mentions | umls-concept:C1705987 | lld:lifeskim |
| pubmed-article:9350434 | lifeskim:mentions | umls-concept:C1314939 | lld:lifeskim |
| pubmed-article:9350434 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:9350434 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:9350434 | pubmed:dateCreated | 1997-12-4 | lld:pubmed |
| pubmed-article:9350434 | pubmed:abstractText | Double-stimulation was used to demonstrate that, in a T lymphocytic cell line (CEM), phorbol myristate acetate (PMA) rapidly induced NF-kappa B through a signaling pathway which did not involve reactive oxygen species (ROS) and was different from the activation triggered by either H2O2 or tumor necrosis factor-alpha (TNF-alpha). Since these latter compounds were known to activate NF-kappa B translocation in a redox-sensitive way, we have demonstrated that NF-kappa B activation by PMA was resistant to antioxidant N-acetyl-L-cysteine (NAC) and sensitive to kinase inhibitors staurosporine and H7 while activation by H2O2 or TNF-alpha were not. | lld:pubmed |
| pubmed-article:9350434 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9350434 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9350434 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:9350434 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9350434 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:9350434 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9350434 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:9350434 | pubmed:issn | 1071-5762 | lld:pubmed |
| pubmed-article:9350434 | pubmed:author | pubmed-author:PietteJJ | lld:pubmed |
| pubmed-article:9350434 | pubmed:author | pubmed-author:PiresMM | lld:pubmed |
| pubmed-article:9350434 | pubmed:author | pubmed-author:SchoonbroodtS... | lld:pubmed |
| pubmed-article:9350434 | pubmed:author | pubmed-author:Legrand-Poels... | lld:pubmed |
| pubmed-article:9350434 | pubmed:author | pubmed-author:ZecchinonLL | lld:pubmed |
| pubmed-article:9350434 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9350434 | pubmed:volume | 27 | lld:pubmed |
| pubmed-article:9350434 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9350434 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9350434 | pubmed:pagination | 301-9 | lld:pubmed |
| pubmed-article:9350434 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:9350434 | pubmed:year | 1997 | lld:pubmed |
| pubmed-article:9350434 | pubmed:articleTitle | Involvement of different transduction pathways in NF-kappa B activation by several inducers. | lld:pubmed |
| pubmed-article:9350434 | pubmed:affiliation | Laboratory of Virology, University of Liège, Belgium. | lld:pubmed |
| pubmed-article:9350434 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9350434 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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