pubmed-article:9317046 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:9317046 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:9317046 | lifeskim:mentions | umls-concept:C0006305 | lld:lifeskim |
pubmed-article:9317046 | lifeskim:mentions | umls-concept:C0024432 | lld:lifeskim |
pubmed-article:9317046 | lifeskim:mentions | umls-concept:C0038952 | lld:lifeskim |
pubmed-article:9317046 | lifeskim:mentions | umls-concept:C0596988 | lld:lifeskim |
pubmed-article:9317046 | lifeskim:mentions | umls-concept:C1442161 | lld:lifeskim |
pubmed-article:9317046 | lifeskim:mentions | umls-concept:C0056922 | lld:lifeskim |
pubmed-article:9317046 | pubmed:issue | 10 | lld:pubmed |
pubmed-article:9317046 | pubmed:dateCreated | 1997-10-30 | lld:pubmed |
pubmed-article:9317046 | pubmed:abstractText | A bacterioferritin (BFR) deletion mutant of Brucella melitensis 16M was generated by gene replacement. The deletion was complemented with a broad-host-range vector carrying the wild-type bfr gene, pBBR-bfr. The survival and growth of the mutant, B. melitensis PAD 2-78, were similar to those of its parental strain in human monocyte-derived macrophages (MDM). These results suggest that BFR is not essential for the intracellular survival of B. melitensis in human MDM. | lld:pubmed |
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pubmed-article:9317046 | pubmed:language | eng | lld:pubmed |
pubmed-article:9317046 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9317046 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:9317046 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:9317046 | pubmed:month | Oct | lld:pubmed |
pubmed-article:9317046 | pubmed:issn | 0019-9567 | lld:pubmed |
pubmed-article:9317046 | pubmed:author | pubmed-author:HooverD LDL | lld:pubmed |
pubmed-article:9317046 | pubmed:author | pubmed-author:LetessonJ JJJ | lld:pubmed |
pubmed-article:9317046 | pubmed:author | pubmed-author:ZygmuntM SMS | lld:pubmed |
pubmed-article:9317046 | pubmed:author | pubmed-author:CrawfordR MRM | lld:pubmed |
pubmed-article:9317046 | pubmed:author | pubmed-author:TiborAA | lld:pubmed |
pubmed-article:9317046 | pubmed:author | pubmed-author:DenoelP APA | lld:pubmed |
pubmed-article:9317046 | pubmed:author | pubmed-author:WeynantsV EVE | lld:pubmed |
pubmed-article:9317046 | pubmed:author | pubmed-author:GodfroidFF | lld:pubmed |
pubmed-article:9317046 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:9317046 | pubmed:volume | 65 | lld:pubmed |
pubmed-article:9317046 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:9317046 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:9317046 | pubmed:pagination | 4337-40 | lld:pubmed |
pubmed-article:9317046 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:9317046 | pubmed:year | 1997 | lld:pubmed |
pubmed-article:9317046 | pubmed:articleTitle | Survival of a bacterioferritin deletion mutant of Brucella melitensis 16M in human monocyte-derived macrophages. | lld:pubmed |
pubmed-article:9317046 | pubmed:affiliation | U. R. Biologie Moléculaire, Laboratoire de Microbiologie et d'Immunologie, Facultés Universitaires Notre Dame de la Paix, Namur, Belgium. Philippe.Denoel@fundp.ac.be | lld:pubmed |
pubmed-article:9317046 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:9317046 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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