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Predicate | Object |
---|---|
rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
|
pubmed:dateCreated |
1997-9-15
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pubmed:abstractText |
Myocardial ischemia is known to upregulate the systemic renin-angiotensin system, which influences myocardial ischemic events by affecting hemodynamics and hemostatic activity. This study was designed to examine whether angiotensin II (Ang II) receptor expression in the myocardium is altered immediately after ischemia-reperfusion.
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pubmed:language |
eng
|
pubmed:journal | |
pubmed:citationSubset |
AIM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Aug
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pubmed:issn |
0009-7322
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:day |
5
|
pubmed:volume |
96
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
|
pubmed:pagination |
922-6
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:9264502-Animals,
pubmed-meshheading:9264502-Autoradiography,
pubmed-meshheading:9264502-Male,
pubmed-meshheading:9264502-Myocardial Ischemia,
pubmed-meshheading:9264502-Myocardial Reperfusion,
pubmed-meshheading:9264502-Myocardium,
pubmed-meshheading:9264502-Rats,
pubmed-meshheading:9264502-Rats, Sprague-Dawley,
pubmed-meshheading:9264502-Receptors, Angiotensin,
pubmed-meshheading:9264502-Time Factors
|
pubmed:year |
1997
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pubmed:articleTitle |
Increase in angiotensin II type 1 receptor expression immediately after ischemia-reperfusion in isolated rat hearts.
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pubmed:affiliation |
Department of Medicine, University of Florida and VA Medical Center, Gainesville 32610, USA.
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pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, U.S. Gov't, Non-P.H.S.,
Research Support, Non-U.S. Gov't
|