Linked Life Data

9236453

Source:http://linkedlifedata.com/resource/pubmed/id/9236453

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
1997-8-14
pubmed:abstractText
Ischemia-reperfusion injury leads to the activation and endothelial deposition of complement. We investigated whether exposure of human umbilical vein endothelial cells (HUVECs) to hypoxia and/or reoxygenation activates complement and decreases HUVEC-surface expression of the C3 regulatory proteins CD46 and CD55.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jul
pubmed:issn
0009-7322
pubmed:author
pubmed:issnType
Print
pubmed:day
1
pubmed:volume
96
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
326-33
pubmed:dateRevised
2007-11-14
pubmed:meshHeading
pubmed:year
1997
pubmed:articleTitle
Reoxygenation of hypoxic human umbilical vein endothelial cells activates the classic complement pathway.
pubmed:affiliation
Department of Anesthesia, Brigham and Women's Hospital, Harvard Medical School, Boston, Mass. 02115, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S.