|
rdf:type |
|
|
lifeskim:mentions |
umls-concept:C0002792,
umls-concept:C0021758,
umls-concept:C0024109,
umls-concept:C0030685,
umls-concept:C0086418,
umls-concept:C0391871,
umls-concept:C0439831,
umls-concept:C0680255,
umls-concept:C1274040,
umls-concept:C1283071,
umls-concept:C1963578
|
|
pubmed:issue |
6
|
|
pubmed:dateCreated |
1997-7-24
|
|
pubmed:abstractText |
Interleukin-4 has been implicated as having numerous roles in the inflammatory responses characteristic of allergic asthma. Interleukin-4 has been shown to be involved in IgE synthesis, upregulation of BCAM-1 on endothelium, and promotion of inflammatory cell infiltration into the airways.
|
|
pubmed:language |
eng
|
|
pubmed:journal |
|
|
pubmed:citationSubset |
IM
|
|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
Jun
|
|
pubmed:issn |
1081-1206
|
|
pubmed:author |
|
|
pubmed:issnType |
Print
|
|
pubmed:volume |
78
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
566-8
|
|
pubmed:dateRevised |
2006-11-15
|
|
pubmed:meshHeading |
|
|
pubmed:year |
1997
|
|
pubmed:articleTitle |
Human lung anaphylaxis results in rapid release of interleukin-4.
|
|
pubmed:affiliation |
Department of Internal Medicine, VA Medical Center, Iowa City, Iowa, USA.
|
|
pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, Non-P.H.S.
|
