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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
32
|
pubmed:dateCreated |
1977-12-29
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pubmed:abstractText |
The rate of acetylation of isoniazid is usually constant in a given subject. The authors report here a case where despite an adjusted initial dose there was a decrease in the rate of acetylation during treatment with INH-rifampicin and ethambutol, without hepatic involvement but accompanied by retrobulbar optic neuritis. The role of ethambutol and/or an overdose of isoniazid are discussed. The technique for its estimation is also described.
|
pubmed:language |
fre
|
pubmed:journal | |
pubmed:citationSubset |
IM
|
pubmed:chemical | |
pubmed:status |
MEDLINE
|
pubmed:month |
Oct
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pubmed:issn |
0301-1518
|
pubmed:author | |
pubmed:issnType |
Print
|
pubmed:day |
1
|
pubmed:volume |
6
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
2869-71
|
pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:917803-Acetylation,
pubmed-meshheading:917803-Drug Therapy, Combination,
pubmed-meshheading:917803-Ethambutol,
pubmed-meshheading:917803-Humans,
pubmed-meshheading:917803-Isoniazid,
pubmed-meshheading:917803-Liver,
pubmed-meshheading:917803-Male,
pubmed-meshheading:917803-Optic Neuritis,
pubmed-meshheading:917803-Rifampin,
pubmed-meshheading:917803-Tuberculosis
|
pubmed:year |
1977
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pubmed:articleTitle |
[Modification during treatment of the rate of acetylation of isoniazide clinically or biologically detectable hepatic damage (author's transl)].
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pubmed:publicationType |
Journal Article,
English Abstract,
Case Reports
|