Linked Life Data

9157370

Source:http://linkedlifedata.com/resource/pubmed/id/9157370

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
1997-5-22
pubmed:abstractText
The purpose of the study was to examine the pattern of changes revealed in the metabolic reactions in the cerebral mitochondria, oxidizing succinates in acute circulatory hypoxia under the influence of hyper- and normoxic conditions of an experiment. Following 3.5 hours of acute circulatory hypoxia in the cerebral mitochondria which were resistant to bilateral carotid occlusion, rats showed increased succinate-dependent energy production, followed by compensatory inhibition of succinate dehydrogenase. The normoxic conditions of the cerebral mitochondrial investigations preserved succinate-dependent and transaminase respiratory fractions of organelles which were inhibited under hyperoxic conditions of an experiment. This improves more objective assessment of the responses of the mitochondrial oxidation system on the pathology model under study.
pubmed:language
rus
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
0031-2991
pubmed:author
pubmed:issnType
Print
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
10-2
pubmed:dateRevised
2006-11-15
pubmed:meshHeading
pubmed:articleTitle
[Effect of research conditions on succinate oxidation in brain mitochondria in circulatory hypoxia].
pubmed:publicationType
Journal Article, English Abstract