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rdf:type |
|
|
lifeskim:mentions |
|
|
pubmed:issue |
4
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|
pubmed:dateCreated |
1997-4-22
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|
pubmed:abstractText |
Paraplegia remains a serious complication of thoracoabdominal aortic operations. However, despite growing in vitro evidence, it has been difficult to demonstrate glutamate neurotoxicity in vivo because of the reuptake activity that occurs. We hypothesized that glutamate can be toxic to the spinal cord under metabolic stress.
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|
pubmed:language |
eng
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|
pubmed:journal |
|
|
pubmed:citationSubset |
AIM
|
|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
Apr
|
|
pubmed:issn |
0003-4975
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|
pubmed:author |
pubmed-author:AebaRR,
pubmed-author:ItoTT,
pubmed-author:KawadaSS,
pubmed-author:KotoAA,
pubmed-author:MitsumaruAA,
pubmed-author:MoriAA,
pubmed-author:NakamichiTT,
pubmed-author:OdaguchiHH,
pubmed-author:UedaTT,
pubmed-author:YasudoMM,
pubmed-author:YozuRR
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|
pubmed:issnType |
Print
|
|
pubmed:volume |
63
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
1057-62
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|
pubmed:dateRevised |
2003-11-14
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|
pubmed:meshHeading |
|
|
pubmed:year |
1997
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|
pubmed:articleTitle |
Detrimental effects of exogenous glutamate on spinal cord neurons during brief ischemia in vivo.
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|
pubmed:affiliation |
Department of Cardiovascular Surgery, Keio University, Shinjuku, Tokyo, Japan.
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|
pubmed:publicationType |
Journal Article
|
