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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
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pubmed:dateCreated |
1997-6-9
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pubmed:abstractText |
The preservation of gastric mucosal homeostasis is a complex biologic process, controlled by a dynamic equilibrium of cell loss by apoptosis with that of cellular proliferation, and its abrogation is a prominent feature of Helicobacter pylori-associated gastritis. In this report, we show that H. pylori lipopolysaccharide induces histologic lesions typical of acute gastritis and that these changes are reflected in the increased epithelial cell apoptosis.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
0036-5521
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
32
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
203-11
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pubmed:dateRevised |
2003-11-14
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pubmed:meshHeading |
pubmed-meshheading:9085455-Animals,
pubmed-meshheading:9085455-Apoptosis,
pubmed-meshheading:9085455-Dose-Response Relationship, Immunologic,
pubmed-meshheading:9085455-Epithelium,
pubmed-meshheading:9085455-Gastric Mucosa,
pubmed-meshheading:9085455-Gastritis,
pubmed-meshheading:9085455-Helicobacter pylori,
pubmed-meshheading:9085455-Lipopolysaccharides,
pubmed-meshheading:9085455-Rats,
pubmed-meshheading:9085455-Rats, Sprague-Dawley
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pubmed:year |
1997
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pubmed:articleTitle |
Induction of acute gastritis and epithelial apoptosis by Helicobacter pylori lipopolysaccharide.
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pubmed:affiliation |
Research Center, University of Medicine and Dentistry of New Jersey, Newark 07103-2400, USA.
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pubmed:publicationType |
Journal Article
|