Linked Life Data

9082788

Source:http://linkedlifedata.com/resource/pubmed/id/9082788

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:dateCreated
1997-4-3
pubmed:abstractText
To define the role of adenotonsillar hypertrophy and facial morphology in obstructive sleep apnea (OSA) in children we performed a cephalometric analysis of an OSA group and an age-matched control group. Adenotonsillar hypertrophy was remarkable in OSA children at every age level. Maxillary protrusion expressed by SNA was significantly smaller in the OSA group than in the control group for elderly children (5-9 years old). Mandibul protrusion expressed by SNB was significantly smaller in the OSA group even at younger ages (1-2 years). The hyoid bone was significantly lower in the OSA group than in the control group who were from 3 to 6 years. Environmental factors due to upper airway obstruction as well as genetic factors are suspected to cause abnormal facial morphology in OSA children.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:status
MEDLINE
pubmed:issn
0365-5237
pubmed:author
pubmed:issnType
Print
pubmed:volume
523
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
222-4
pubmed:dateRevised
2008-2-13
pubmed:meshHeading
pubmed:year
1996
pubmed:articleTitle
Adenotonsillar hypertrophy and skeletal morphology of children with obstructive sleep apnea syndrome.
pubmed:affiliation
Department of Otolaryngology, Sapporo Medical University, Sapporo, Japan.
pubmed:publicationType
Journal Article