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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
|
pubmed:dateCreated |
1997-4-10
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pubmed:abstractText |
Recent findings of a reduced cerebral metabolic rate of glucose (CMRGlu) in at-risk relatives of patients with Alzheimer disease (AD) who carry the apolipoprotein E (APOE) epsilon 4 allele suggest a causative role for the E4 isoform in cognitive changes that lead to AD. It is not known whether epsilon 4 allele-associated deficits exist in patients with clinical AD.
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pubmed:language |
eng
|
pubmed:journal | |
pubmed:citationSubset |
AIM
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pubmed:chemical | |
pubmed:status |
MEDLINE
|
pubmed:month |
Mar
|
pubmed:issn |
0003-9942
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pubmed:author | |
pubmed:issnType |
Print
|
pubmed:volume |
54
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pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
273-7
|
pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:9074396-Adult,
pubmed-meshheading:9074396-Aged,
pubmed-meshheading:9074396-Alleles,
pubmed-meshheading:9074396-Alzheimer Disease,
pubmed-meshheading:9074396-Apolipoproteins E,
pubmed-meshheading:9074396-Brain,
pubmed-meshheading:9074396-Genotype,
pubmed-meshheading:9074396-Glucose,
pubmed-meshheading:9074396-Humans,
pubmed-meshheading:9074396-Middle Aged
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pubmed:year |
1997
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pubmed:articleTitle |
No difference in cerebral glucose metabolism in patients with Alzheimer disease and differing apolipoprotein E genotypes.
|
pubmed:affiliation |
Department of Clinical Neuroscience and Family Medicine, Karolinska Institute, Huddinge University Hospital, Sweden.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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