pubmed-article:9054857 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:9054857 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
pubmed-article:9054857 | lifeskim:mentions | umls-concept:C0003507 | lld:lifeskim |
pubmed-article:9054857 | lifeskim:mentions | umls-concept:C0149721 | lld:lifeskim |
pubmed-article:9054857 | lifeskim:mentions | umls-concept:C0027096 | lld:lifeskim |
pubmed-article:9054857 | lifeskim:mentions | umls-concept:C0597298 | lld:lifeskim |
pubmed-article:9054857 | lifeskim:mentions | umls-concept:C0086168 | lld:lifeskim |
pubmed-article:9054857 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
pubmed-article:9054857 | lifeskim:mentions | umls-concept:C1420192 | lld:lifeskim |
pubmed-article:9054857 | lifeskim:mentions | umls-concept:C1707719 | lld:lifeskim |
pubmed-article:9054857 | lifeskim:mentions | umls-concept:C0205385 | lld:lifeskim |
pubmed-article:9054857 | lifeskim:mentions | umls-concept:C0027119 | lld:lifeskim |
pubmed-article:9054857 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:9054857 | pubmed:dateCreated | 1997-3-28 | lld:pubmed |
pubmed-article:9054857 | pubmed:abstractText | Reexpression of the fetal beta-myosin heavy chain (beta-MHC) gene was reported to be a marker for phenotypic reprogramming and cardiac hypertrophy in rats. Recent in vitro studies strongly suggested a role of angiotensin II for phenotypic reprogramming. In the present investigation, beta-MHC gene expression was studied in an experimental model of pressure-over-load hypertrophy that is not associated with a concurrent activation of the circulating renin-angiotensin system. | lld:pubmed |
pubmed-article:9054857 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9054857 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9054857 | pubmed:language | eng | lld:pubmed |
pubmed-article:9054857 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9054857 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:9054857 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9054857 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9054857 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:9054857 | pubmed:month | Mar | lld:pubmed |
pubmed-article:9054857 | pubmed:issn | 0009-7322 | lld:pubmed |
pubmed-article:9054857 | pubmed:author | pubmed-author:ZakRR | lld:pubmed |
pubmed-article:9054857 | pubmed:author | pubmed-author:RüeggJ CJC | lld:pubmed |
pubmed-article:9054857 | pubmed:author | pubmed-author:WiesnerR JRJ | lld:pubmed |
pubmed-article:9054857 | pubmed:author | pubmed-author:EhmkeHH | lld:pubmed |
pubmed-article:9054857 | pubmed:author | pubmed-author:FaulhaberJJ | lld:pubmed |
pubmed-article:9054857 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:9054857 | pubmed:day | 4 | lld:pubmed |
pubmed-article:9054857 | pubmed:volume | 95 | lld:pubmed |
pubmed-article:9054857 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:9054857 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:9054857 | pubmed:pagination | 1253-9 | lld:pubmed |
pubmed-article:9054857 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
pubmed-article:9054857 | pubmed:meshHeading | pubmed-meshheading:9054857-... | lld:pubmed |
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pubmed-article:9054857 | pubmed:meshHeading | pubmed-meshheading:9054857-... | lld:pubmed |
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pubmed-article:9054857 | pubmed:meshHeading | pubmed-meshheading:9054857-... | lld:pubmed |
pubmed-article:9054857 | pubmed:meshHeading | pubmed-meshheading:9054857-... | lld:pubmed |
pubmed-article:9054857 | pubmed:meshHeading | pubmed-meshheading:9054857-... | lld:pubmed |
pubmed-article:9054857 | pubmed:meshHeading | pubmed-meshheading:9054857-... | lld:pubmed |
pubmed-article:9054857 | pubmed:meshHeading | pubmed-meshheading:9054857-... | lld:pubmed |
pubmed-article:9054857 | pubmed:meshHeading | pubmed-meshheading:9054857-... | lld:pubmed |
pubmed-article:9054857 | pubmed:year | 1997 | lld:pubmed |
pubmed-article:9054857 | pubmed:articleTitle | Dissociation of left ventricular hypertrophy, beta-myosin heavy chain gene expression, and myosin isoform switch in rats after ascending aortic stenosis. | lld:pubmed |
pubmed-article:9054857 | pubmed:affiliation | Department of Physiology, University of Heidelberg, Germany. rudolf.wiesner@urz.uni-heidelberg.de | lld:pubmed |
pubmed-article:9054857 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:9054857 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:9054857 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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