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rdf:type |
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lifeskim:mentions |
umls-concept:C0003507,
umls-concept:C0017262,
umls-concept:C0027096,
umls-concept:C0027119,
umls-concept:C0034693,
umls-concept:C0086168,
umls-concept:C0149721,
umls-concept:C0205385,
umls-concept:C0597298,
umls-concept:C1420192,
umls-concept:C1707719
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pubmed:issue |
5
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|
pubmed:dateCreated |
1997-3-28
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pubmed:abstractText |
Reexpression of the fetal beta-myosin heavy chain (beta-MHC) gene was reported to be a marker for phenotypic reprogramming and cardiac hypertrophy in rats. Recent in vitro studies strongly suggested a role of angiotensin II for phenotypic reprogramming. In the present investigation, beta-MHC gene expression was studied in an experimental model of pressure-over-load hypertrophy that is not associated with a concurrent activation of the circulating renin-angiotensin system.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
0009-7322
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:day |
4
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pubmed:volume |
95
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1253-9
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:9054857-Analysis of Variance,
pubmed-meshheading:9054857-Animals,
pubmed-meshheading:9054857-Aorta, Abdominal,
pubmed-meshheading:9054857-Aorta, Thoracic,
pubmed-meshheading:9054857-Aortic Valve Stenosis,
pubmed-meshheading:9054857-Cardiomegaly,
pubmed-meshheading:9054857-Female,
pubmed-meshheading:9054857-Heart Ventricles,
pubmed-meshheading:9054857-Hemodynamics,
pubmed-meshheading:9054857-Hypertrophy, Left Ventricular,
pubmed-meshheading:9054857-Myocardium,
pubmed-meshheading:9054857-Myosin Heavy Chains,
pubmed-meshheading:9054857-Polymerase Chain Reaction,
pubmed-meshheading:9054857-RNA, Messenger,
pubmed-meshheading:9054857-Rats,
pubmed-meshheading:9054857-Rats, Sprague-Dawley,
pubmed-meshheading:9054857-Transcription, Genetic
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pubmed:year |
1997
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pubmed:articleTitle |
Dissociation of left ventricular hypertrophy, beta-myosin heavy chain gene expression, and myosin isoform switch in rats after ascending aortic stenosis.
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pubmed:affiliation |
Department of Physiology, University of Heidelberg, Germany. rudolf.wiesner@urz.uni-heidelberg.de
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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