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pubmed-article:9021210 | lifeskim:mentions | umls-concept:C0036957 | lld:lifeskim |
pubmed-article:9021210 | lifeskim:mentions | umls-concept:C0678226 | lld:lifeskim |
pubmed-article:9021210 | lifeskim:mentions | umls-concept:C0162735 | lld:lifeskim |
pubmed-article:9021210 | lifeskim:mentions | umls-concept:C2924612 | lld:lifeskim |
pubmed-article:9021210 | lifeskim:mentions | umls-concept:C0179400 | lld:lifeskim |
pubmed-article:9021210 | lifeskim:mentions | umls-concept:C0163786 | lld:lifeskim |
pubmed-article:9021210 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:9021210 | pubmed:dateCreated | 1997-5-5 | lld:pubmed |
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pubmed-article:9021210 | pubmed:abstractText | TEM-1 hyperproduction in two ampicillin-sulbactam-resistant Shigella flexneri strains was studied. In both strains the blaTEM gene was encoded as a single copy on a large conjugatively transferable plasmid. A single G-->T transversion at position 1 of the -10 consensus sequence was identified to be the mechanism of TEM-1 hyperproduction. | lld:pubmed |
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pubmed-article:9021210 | pubmed:language | eng | lld:pubmed |
pubmed-article:9021210 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:9021210 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:9021210 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:9021210 | pubmed:month | Feb | lld:pubmed |
pubmed-article:9021210 | pubmed:issn | 0066-4804 | lld:pubmed |
pubmed-article:9021210 | pubmed:author | pubmed-author:ChauP YPY | lld:pubmed |
pubmed-article:9021210 | pubmed:author | pubmed-author:WongS SSS | lld:pubmed |
pubmed-article:9021210 | pubmed:author | pubmed-author:HoP LPL | lld:pubmed |
pubmed-article:9021210 | pubmed:author | pubmed-author:SinL DLD | lld:pubmed |
pubmed-article:9021210 | pubmed:author | pubmed-author:YuenK YKY | lld:pubmed |
pubmed-article:9021210 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:9021210 | pubmed:volume | 41 | lld:pubmed |
pubmed-article:9021210 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:9021210 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:9021210 | pubmed:pagination | 468-70 | lld:pubmed |
pubmed-article:9021210 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:9021210 | pubmed:year | 1997 | lld:pubmed |
pubmed-article:9021210 | pubmed:articleTitle | Transferable hyperproduction of TEM-1 beta-lactamase in Shigella flexneri due to a point mutation in the pribnow box. | lld:pubmed |
pubmed-article:9021210 | pubmed:affiliation | Department of Microbiology, Queen Mary Hospital Compound, University of Hong Kong, Hong Kong. | lld:pubmed |
pubmed-article:9021210 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:9021210 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:9021210 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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