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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
12
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pubmed:dateCreated |
1997-2-3
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pubmed:abstractText |
Antibodies to selected neutrophil or endothelial cell adhesion molecules decrease myocardial infarct size in vivo. Platelet/endothelial cell adhesion molecule-1 (PECAM-1) is an immunoglobulin gene superfamily member expressed constitutively on neutrophils and endothelium. F(ab')2 fragments of antibody against PECAM-1 inhibit transendothelial migration of neutrophils in several in vivo models of acute inflammation. Therefore, we examined the effect of F(ab')2 fragments of anti-PECAM-1 antibody in a rat model of myocardial infarction.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
AIM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
0009-7322
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:day |
15
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pubmed:volume |
94
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
3327-33
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:8989147-Analysis of Variance,
pubmed-meshheading:8989147-Animals,
pubmed-meshheading:8989147-Antigens, CD31,
pubmed-meshheading:8989147-Hemodynamics,
pubmed-meshheading:8989147-Humans,
pubmed-meshheading:8989147-Immunoglobulin Fab Fragments,
pubmed-meshheading:8989147-Immunoglobulin G,
pubmed-meshheading:8989147-Leukocytes,
pubmed-meshheading:8989147-Male,
pubmed-meshheading:8989147-Myocardial Infarction,
pubmed-meshheading:8989147-Myocardial Reperfusion Injury,
pubmed-meshheading:8989147-Peroxidase,
pubmed-meshheading:8989147-Rabbits,
pubmed-meshheading:8989147-Rats,
pubmed-meshheading:8989147-Rats, Wistar
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pubmed:year |
1996
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pubmed:articleTitle |
Antibody to platelet/endothelial cell adhesion molecule-1 reduces myocardial infarct size in a rat model of ischemia-reperfusion injury.
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pubmed:affiliation |
Medical College of Wisconsin, Department of Cellular Biology and Anatomy, Milwaukee 53226, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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