| pubmed-article:8989110 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8989110 | lifeskim:mentions | umls-concept:C0042373 | lld:lifeskim |
| pubmed-article:8989110 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:8989110 | lifeskim:mentions | umls-concept:C0066357 | lld:lifeskim |
| pubmed-article:8989110 | lifeskim:mentions | umls-concept:C1327009 | lld:lifeskim |
| pubmed-article:8989110 | lifeskim:mentions | umls-concept:C1707520 | lld:lifeskim |
| pubmed-article:8989110 | lifeskim:mentions | umls-concept:C0205214 | lld:lifeskim |
| pubmed-article:8989110 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:8989110 | pubmed:dateCreated | 1997-2-3 | lld:pubmed |
| pubmed-article:8989110 | pubmed:abstractText | Increased homocysteine levels are a risk factor for atherosclerosis and its sequelae. A common genetic mutation in methylenetetrahydrofolate reductase (MTHFR), an enzyme required for efficient homocysteine metabolism, creates a thermolabile enzyme with reduced activity. We determined the prevalence of this mutation in many subjects with and without vascular disease and related it to homocysteine and folate levels. | lld:pubmed |
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| pubmed-article:8989110 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8989110 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8989110 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8989110 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:8989110 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8989110 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:8989110 | pubmed:issn | 0009-7322 | lld:pubmed |
| pubmed-article:8989110 | pubmed:author | pubmed-author:EvansAA | lld:pubmed |
| pubmed-article:8989110 | pubmed:author | pubmed-author:McWilliamsJJ | lld:pubmed |
| pubmed-article:8989110 | pubmed:author | pubmed-author:SadeghiAA | lld:pubmed |
| pubmed-article:8989110 | pubmed:author | pubmed-author:TaylorL MLMJr | lld:pubmed |
| pubmed-article:8989110 | pubmed:author | pubmed-author:PressR DRD | lld:pubmed |
| pubmed-article:8989110 | pubmed:author | pubmed-author:HennerW DWD | lld:pubmed |
| pubmed-article:8989110 | pubmed:author | pubmed-author:DelougheryT... | lld:pubmed |
| pubmed-article:8989110 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8989110 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:8989110 | pubmed:volume | 94 | lld:pubmed |
| pubmed-article:8989110 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8989110 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8989110 | pubmed:pagination | 3074-8 | lld:pubmed |
| pubmed-article:8989110 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:8989110 | pubmed:year | 1996 | lld:pubmed |
| pubmed-article:8989110 | pubmed:articleTitle | Common mutation in methylenetetrahydrofolate reductase. Correlation with homocysteine metabolism and late-onset vascular disease. | lld:pubmed |
| pubmed-article:8989110 | pubmed:affiliation | Department of Medicine, Oregon Health Sciences University, Portland 97201-3098, USA. delought@ccmail.ohsu.edu | lld:pubmed |
| pubmed-article:8989110 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8989110 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:8989110 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:8989110 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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